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J Thorac Dis. 2019 Jan;11(1):47-49. doi: 10.21037/jtd.2018.12.117.
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The role of epithelial to mesenchymal transition in resistance to epidermal growth factor receptor tyrosine kinase inhibitors in non-small cell lung cancer.上皮间质转化在非小细胞肺癌表皮生长因子受体酪氨酸激酶抑制剂耐药中的作用。
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[Research Progress of the Role of EMT in EGFR-TKIs Resistance 
of Non-small Cell Lung Cancer].[上皮-间质转化在非小细胞肺癌表皮生长因子受体酪氨酸激酶抑制剂耐药中作用的研究进展]
Zhongguo Fei Ai Za Zhi. 2018 Dec 20;21(12):907-911. doi: 10.3779/j.issn.1009-3419.2018.12.08.
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Epithelial-to-mesenchymal transition and its role in EGFR-mutant lung adenocarcinoma and idiopathic pulmonary fibrosis.上皮-间质转化及其在表皮生长因子受体突变型肺腺癌和特发性肺纤维化中的作用。
Pathol Int. 2017 Aug;67(8):379-388. doi: 10.1111/pin.12553. Epub 2017 Jul 5.

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The pan-cancer landscape of crosstalk between epithelial-mesenchymal transition and immune evasion relevant to prognosis and immunotherapy response.与预后和免疫治疗反应相关的上皮-间质转化与免疫逃逸之间串扰的泛癌格局。
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Epithelial-to-mesenchymal transition and drug resistance: transitioning away from death.上皮-间质转化与耐药性：摆脱死亡的转变
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本文引用的文献

1
Targeting the EMT transcription factor TWIST1 overcomes resistance to EGFR inhibitors in EGFR-mutant non-small-cell lung cancer.靶向 EMT 转录因子 TWIST1 克服 EGFR 突变型非小细胞肺癌对 EGFR 抑制剂的耐药性。
Oncogene. 2019 Jan;38(5):656-670. doi: 10.1038/s41388-018-0482-y. Epub 2018 Aug 31.
2
Distinguishing mechanisms underlying EMT tristability.区分上皮-间质转化三稳态背后的机制。
Cancer Converg. 2017;1(1):2. doi: 10.1186/s41236-017-0005-8. Epub 2017 Nov 1.
3
Epithelial-to-Mesenchymal Transition Antagonizes Response to Targeted Therapies in Lung Cancer by Suppressing BIM.上皮-间充质转化通过抑制 BIM 拮抗肺癌对靶向治疗的反应。
Clin Cancer Res. 2018 Jan 1;24(1):197-208. doi: 10.1158/1078-0432.CCR-17-1577. Epub 2017 Oct 19.
4
Epithelial-to-Mesenchymal Transition and MicroRNAs in Lung Cancer.肺癌中的上皮-间质转化与微小RNA
Cancers (Basel). 2017 Aug 3;9(8):101. doi: 10.3390/cancers9080101.
5
Therapeutic Targeting of Epithelial Plasticity Programs: Focus on the Epithelial-Mesenchymal Transition.上皮可塑性程序的治疗靶向：聚焦上皮-间质转化
Cells Tissues Organs. 2017;203(2):114-127. doi: 10.1159/000447238. Epub 2017 Feb 20.
6
Targeting the miR-200c/LIN28B axis in acquired EGFR-TKI resistance non-small cell lung cancer cells harboring EMT features.针对具有 EMT 特征的获得性 EGFR-TKI 耐药非小细胞肺癌细胞中的 miR-200c/LIN28B 轴。
Sci Rep. 2017 Jan 13;7:40847. doi: 10.1038/srep40847.
7
Combining Anti-Mir-155 with Chemotherapy for the Treatment of Lung Cancers.联合抗微小RNA-155与化疗治疗肺癌
Clin Cancer Res. 2017 Jun 1;23(11):2891-2904. doi: 10.1158/1078-0432.CCR-16-1025. Epub 2016 Nov 30.
8
Tumor Budding: The Name is EMT. Partial EMT.肿瘤芽生：其本质是上皮-间质转化。部分上皮-间质转化。
J Clin Med. 2016 Apr 29;5(5):51. doi: 10.3390/jcm5050051.
9
O-GlcNAc transferase inhibitors: current tools and future challenges.O-连接的N-乙酰葡糖胺转移酶抑制剂：当前工具与未来挑战
Biochem Soc Trans. 2016 Feb;44(1):88-93. doi: 10.1042/BST20150189.
10
PRMT1 Is a Novel Regulator of Epithelial-Mesenchymal-Transition in Non-small Cell Lung Cancer.PRMT1是非小细胞肺癌上皮-间质转化的新型调节因子。
J Biol Chem. 2015 May 22;290(21):13479-89. doi: 10.1074/jbc.M114.636050. Epub 2015 Apr 6.

How apoptosis and epithelial-to-mesenchymal transition are nested in EGFR inhibitors resistance in lung cancer.

作者信息

Garinet Simon, Didelot Audrey, Garelli Elena, Pallier Karine, Blons Hélène, Legras Antoine

机构信息

INSERM UMR-S1147, CNRS SNC 5014, Saints-Pères Research Center, 45 rue des Saints-Pères Paris-Descartes University, Sorbonne Paris Cité University, Paris 75006, France.

Molecular Biology Department, Georges Pompidou European Hospital, Assistance Publique - Hôpitaux de Paris, Paris 75015, France.

出版信息

J Thorac Dis. 2019 Jan;11(1):47-49. doi: 10.21037/jtd.2018.12.117.

DOI:10.21037/jtd.2018.12.117
PMID:30863568
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC6384333/
Abstract
摘要