Demir Bilal, Ozkul Baris, Lapcin Osman, Beng Kubilay, Arikan Yavuz, Yalcinkaya Merter
Department of Orthopaedic Surgery and Traumatology, Metin Sabanci Baltalimani Bone Diseases Training and Research Hospital, Istanbul, Turkey.
Indian J Orthop. 2019 Jan-Feb;53(1):196-203. doi: 10.4103/ortho.IJOrtho_52_17.
Defects of bone and soft tissue occur frequently after high-energy trauma, infections, and tumor resection. Treatment options are limited and outcomes are controversial in nonunion. Classical reconstruction methods are challenging. We describe a method of internal bone transport for treatment of complicated nonunion of the forearm. This method permits axial and internal bone transport without harming the distorted and complex neurovascular anatomy or soft-tissue envelope.
Five patients (mean age, 27 years) with defect nonunion (3 ulna, 2 radius) were treated. Mean preoperative defect size was 36 mm, mean shortening was 14 (0-30) mm, and the extent of surgical resection was 24 (20-40) mm. Total bone loss due to defect, resection, or shortening was 74 mm. According to Paley classification, two of the patients had B1, and three had B3 defect nonunion. This study treats defect nonunion of the forearm using an internal bone-transport method. Our method involved cannulated screws, a cerclage wire, and a circular fixator being used in combination. When transportation was completed, internal fixation of the docking site with a plate and screws was done, with bone grafting after fixator removal. Bone healing and functional outcomes were assessed with radiographs and disabilities of the arm, shoulder, and hand (DASH) scores, respectively.
Mean followup was 67.6 months. Solid osseous union and functional improvement were achieved in all cases. Mean bone loss was 66 mm, mean fixator time was 131.8 days, the lengthening index was 1.3 days/mm, and the fixator index was 2.1 days/mm. DASH score was 82.2 before treatment and 15.36 after treatment.
Using our method, internal bone transport and progressive axial docking of defects can be done with minimal effects on surrounding neurovascular arrangements and soft tissues. Size of fixators can be decreased and formation of painful scar tissue can be avoided.
高能创伤、感染及肿瘤切除术后常出现骨与软组织缺损。治疗选择有限,骨不连的治疗效果存在争议。传统重建方法具有挑战性。我们描述一种用于治疗前臂复杂骨不连的内骨转移方法。该方法允许轴向和内骨转移,而不损伤扭曲复杂的神经血管解剖结构或软组织包膜。
治疗5例(平均年龄27岁)缺损性骨不连患者(尺骨3例,桡骨2例)。术前平均缺损大小为36mm,平均短缩为14(0 - 30)mm,手术切除范围为24(20 - 40)mm。因缺损、切除或短缩导致的总骨丢失为74mm。根据帕利分类,2例患者为B1型,3例为B3型缺损性骨不连。本研究采用内骨转移方法治疗前臂缺损性骨不连。我们的方法是联合使用空心螺钉、环扎钢丝和环形固定器。转移完成后,用钢板和螺钉对对接部位进行内固定,去除固定器后进行植骨。分别通过X线片和手臂、肩部和手部功能障碍(DASH)评分评估骨愈合和功能结果。
平均随访67.6个月。所有病例均实现了牢固的骨愈合和功能改善。平均骨丢失为66mm,平均固定器使用时间为131.8天,延长指数为1.3天/mm,固定器指数为2.1天/mm。治疗前DASH评分为82.2,治疗后为15.36。
采用我们的方法,可进行内骨转移和缺损的渐进性轴向对接,对周围神经血管结构和软组织影响最小。可减小固定器尺寸,避免形成疼痛性瘢痕组织。