Solti F, Szabó Z, Sárai K, Czakó E, Rényi-Vámos F
Acta Chir Acad Sci Hung. 1977;18(4):347-57.
Experimental supraventricular tachyarrhythmia was produced in dogs by the local application of aconitine on the left auricle. The effect of various types of stimulations (right auricular, right ventricular, double-delayed right auricular and right ventricular-right auricular double-delayed stimulation) was studied for the suppression of supra-ventricular (overdrive) tachyarrhythmia. It was found that the electric stimulation of the heart is an effective method of suppressing tachyarrhythmias, though there is a significant difference in the anti-arrhythmic effects of the electric stimulations of various types. For suppression of the supraventricular tachyarrhythmia caused by aconitine, delayed double-right auricular stimulation seemed to be the most effective with respect to the right ventricle. The bioelectric features of aconitine arrhythmia and the mechanism of the antiarrhythmic effect of the electric stimulation of the heart are discussed.
通过在犬的左心耳局部应用乌头碱来诱发实验性室上性快速心律失常。研究了各种类型刺激（右心耳、右心室、双延迟右心耳以及右心室 - 右心耳双延迟刺激）对室上性（超速）快速心律失常的抑制作用。结果发现，心脏电刺激是抑制快速心律失常的有效方法，不过不同类型电刺激的抗心律失常效果存在显著差异。对于抑制由乌头碱引起的室上性快速心律失常而言，相对于右心室，延迟双右心耳刺激似乎最为有效。文中还讨论了乌头碱性心律失常的生物电特征以及心脏电刺激抗心律失常作用的机制。
