Labmayr Viktor, Aliabadi Aryan, Tiesenhausen Kurt, Brodmann Marianne, Schmid Florian, Moore Dana
Department of Orthopaedics and Trauma, University Hospital Graz, Austria.
Division of Angiology, Department of Internal Medicine, University Hospital Graz, Austria.
Case Rep Vasc Med. 2019 Mar 11;2019:8540631. doi: 10.1155/2019/8540631. eCollection 2019.
Popliteal artery entrapment syndrome (PAES) is caused by compression of the popliteal artery (PA) due to deranged myotendinous structures. It can be asymptomatic or may present with exercise intolerance, claudication, or even limb-threatening ischemia. The clinical picture depends on the anatomy and degree of vascular compromise.
We report a case of a 17-year-old Caucasian male with PAES Type II presenting with intermittent claudication and progression towards acute limb ischemia.
MRI and MRA helped identifying the aberrant anatomy and thrombotic occlusion. Doppler ultrasound and conventional angiography have also been employed in a stepwise approach.
The thrombus at the site of occlusion was removed by the use of catheter-directed lysis. Subsequently, popliteal artery release was achieved by myotomy of the aberrant medial head of gastrocnemius muscle (MHGM) and muscle transfer to the medial femoral condyle. A three-month regimen of 60mg edoxaban was recommended after surgery.
Surgical correction of the anomalous anatomy and postoperative anticoagulation led to freedom of symptoms.
Clinical presentation of PAES mimicking peripheral artery occlusive disease is very rare but potentially limb-threatening. PAES should be considered in young and otherwise healthy individuals.
腘动脉受压综合征(PAES)是由肌肌腱结构紊乱导致腘动脉(PA)受压引起的。它可以无症状,也可能表现为运动不耐受、间歇性跛行,甚至危及肢体的缺血。临床表现取决于血管受损的解剖结构和程度。
我们报告一例17岁的白种男性II型PAES患者,表现为间歇性跛行并进展为急性肢体缺血。
MRI和MRA有助于识别异常解剖结构和血栓闭塞。多普勒超声和传统血管造影也已逐步应用。
通过导管定向溶栓清除闭塞部位的血栓。随后,通过对异常的腓肠肌内侧头(MHGM)进行肌切开术并将肌肉转移至股骨内侧髁来实现腘动脉松解。术后推荐使用三个月的60mg依度沙班治疗方案。
异常解剖结构的手术矫正和术后抗凝治疗使症状消失。
PAES模仿外周动脉闭塞性疾病的临床表现非常罕见,但可能危及肢体。对于年轻且其他方面健康的个体应考虑PAES。