Mori Kentaro, Tamada Kota, Kurooka Hisanori, Matsui Makoto, Takumi Toru, Yokota Yoshifumi
Department of Neurology, Kanazawa Medical University, 1-1 Uchinada, Ishikawa 920-0293, Japan.
Division of Molecular Genetics, Department of Biochemistry and Bioinformative Sciences, School of Medicine, Faculty of Medical Sciences, University of Fukui, 23-3 Matsuoka-Shimoaizuki, Eiheiji, Fukui 910-1193, Japan.
Data Brief. 2019 Mar 15;24:103717. doi: 10.1016/j.dib.2019.103717. eCollection 2019 Jun.
This article contains data related to the research article entitled "Id2 determines intestinal identity through repression of the foregut transcription factor, Irx5" [1]. deficient ( ) mice developed gastric tumors and heterotopic squamous epithelium in the small intestine. These tumors and heterotopic tissues were derived from ectopic gastric cells and squamous cells formed in the small intestine respectively during development. In this study, microarray data of the developing small intestine of mice was analyzed.
本文包含与题为“Id2通过抑制前肠转录因子Irx5决定肠道特性”的研究文章相关的数据[1]。Id2缺陷( )小鼠在小肠中出现胃肿瘤和异位鳞状上皮。这些肿瘤和异位组织分别源自发育过程中在小肠中形成的异位胃细胞和鳞状细胞。在本研究中,分析了Id2缺陷小鼠发育中小肠的微阵列数据。