Svendgaard N A, Delgado T J, Arbab M A
Acta Physiol Scand Suppl. 1986;552:90-3.
Subarachnoid blood does not induce vasospasm by a direct vascular effect, but via a neural and/or neurohumoral mechanism. Neurons in the A1 and A2 nuclei and in the median eminence region connected by fibres running in the central tegmental tract are necessary for the development of vasospasm following a SAH.
蛛网膜下腔出血并非通过直接的血管效应诱导血管痉挛,而是经由神经和/或神经体液机制。A1和A2核以及通过中央被盖束纤维相连的正中隆起区域的神经元,对于蛛网膜下腔出血后血管痉挛的发生是必需的。