Respiratory virus infection triggers acute psoriasis flares across different clinical subtypes and genetic backgrounds.
作者信息
Sbidian E, Madrange M, Viguier M, Salmona M, Duchatelet S, Hovnanian A, Smahi A, Le Goff J, Bachelez H
机构信息
AP-HP, Hôpitaux Universitaires Henri Mondor, Département de Dermatologie, UPEC, Créteil, F-94010, France.
Inserm, Centre d'Investigation Clinique 1430, Créteil, F-94010, France.
出版信息
Br J Dermatol. 2019 Dec;181(6):1304-1306. doi: 10.1111/bjd.18203. Epub 2019 Aug 20.
Abstract
摘要
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IL36RN Mutations Affect Protein Expression and Function: A Basis for Genotype-Phenotype Correlation in Pustular Diseases.IL36RN突变影响蛋白质表达和功能：脓疱性疾病中基因型-表型相关性的基础。
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Psoriasis.银屑病。
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Interleukin-36-receptor antagonist deficiency and generalized pustular psoriasis.白细胞介素-36 受体拮抗剂缺乏与泛发性脓疱型银屑病。
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HIV-associated psoriasis: pathogenesis, clinical features, and management.HIV 相关性银屑病：发病机制、临床特征及治疗。
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Toll-like receptor 3 is induced by and mediates antiviral activity against rhinovirus infection of human bronchial epithelial cells.Toll样受体3由人支气管上皮细胞的鼻病毒感染诱导产生，并介导针对该感染的抗病毒活性。
J Virol. 2005 Oct;79(19):12273-9. doi: 10.1128/JVI.79.19.12273-12279.2005.
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