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Mineralocorticoid antagonists and ENaC inhibitors in hyperaldosteronism.

作者信息

Hill Michael A, Sowers James R

机构信息

Diabetes and Cardiovascular Center, Dalton Cardiovascular Research Center, VA Medical Center, University of Missouri School of Medicine, Columbia, Missouri.

出版信息

J Clin Hypertens (Greenwich). 2019 Jul;21(7):929-931. doi: 10.1111/jch.13566. Epub 2019 Jun 6.

DOI:10.1111/jch.13566
PMID:31169977
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8030310/
Abstract
摘要

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Mineralocorticoid antagonists and ENaC inhibitors in hyperaldosteronism.原发性醛固酮增多症中的盐皮质激素拮抗剂和上皮钠通道抑制剂
J Clin Hypertens (Greenwich). 2019 Jul;21(7):929-931. doi: 10.1111/jch.13566. Epub 2019 Jun 6.
2
Blood pressure responses to small doses of amiloride and spironolactone in normotensive subjects.正常血压受试者对小剂量阿米洛利和螺内酯的血压反应。
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3
[TREATMENT OF STATES OF HYPERALDOSTERONISM. THERAPEUTIC INDICATIONS OF SPIRONOLACTONE].
Rev Prat. 1963 Sep 21;13:2962-75.
4
SFE/SFHTA/AFCE consensus on primary aldosteronism, part 7: Medical treatment of primary aldosteronism.原发性醛固酮增多症的 SFE/SFHTA/AFCE 共识，第 7 部分：原发性醛固酮增多症的药物治疗。
Ann Endocrinol (Paris). 2016 Jul;77(3):226-34. doi: 10.1016/j.ando.2016.01.010. Epub 2016 Jun 14.
5
Comparison of agents that affect aldosterone action.影响醛固酮作用的药物比较。
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6
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Eur J Clin Pharmacol. 2020 Jan;76(1):131-132. doi: 10.1007/s00228-019-02774-2. Epub 2019 Oct 29.
8
[Physiological bases of the utilization of aldosterone antagonists. Their clinical applications].
Acta Cardiol. 1962;17:627-56.
9
[The hyperaldosteronism syndrome in edema caused by decompensated respiratory insufficiency and its treatment by spirolactones].
Poumon Coeur. 1962;18:747-54.
10
[SECONDARY HYPERALDOSTERONISM. PHYSIOPATHOLOGIC, CLINICAL AND THERAPEUTIC CONSIDERATIONS].[继发性醛固酮增多症。病理生理、临床及治疗方面的考量]
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本文引用的文献

1
Long-term BP control and vascular health in patients with hyperaldosteronism treated with low-dose, amiloride-based therapy.低剂量阿米洛利为基础治疗的醛固酮增多症患者的长期血压控制和血管健康。
J Clin Hypertens (Greenwich). 2019 Jul;21(7):922-928. doi: 10.1111/jch.13567. Epub 2019 Jun 6.
2
Multifaceted Functions of Epithelial Na Channel in Modulating Blood Pressure.上皮钠通道在调节血压中的多方面功能
Hypertension. 2019 Feb;73(2):273-281. doi: 10.1161/HYPERTENSIONAHA.118.12330.
3
Role of Renin-Angiotensin-Aldosterone System Activation in Promoting Cardiovascular Fibrosis and Stiffness.肾素-血管紧张素-醛固酮系统激活在促进心血管纤维化和僵硬中的作用。
Hypertension. 2018 Sep;72(3):537-548. doi: 10.1161/HYPERTENSIONAHA.118.11065.
4
Epithelial Sodium Channel in Aldosterone-Induced Endothelium Stiffness and Aortic Dysfunction.醛固酮诱导的血管内皮僵硬和主动脉功能障碍中的上皮钠通道。
Hypertension. 2018 Sep;72(3):731-738. doi: 10.1161/HYPERTENSIONAHA.118.11339.
5
Potential Role of Antihypertensive Medications in Preventing Excessive Arterial Stiffening.抗高血压药物在预防动脉过度僵硬中的潜在作用。
Curr Hypertens Rep. 2018 Jul 6;20(9):76. doi: 10.1007/s11906-018-0876-9.
6
Cardiovascular events and target organ damage in primary aldosteronism compared with essential hypertension: a systematic review and meta-analysis.原发性醛固酮增多症与原发性高血压的心血管事件和靶器官损害的比较：系统评价和荟萃分析。
Lancet Diabetes Endocrinol. 2018 Jan;6(1):41-50. doi: 10.1016/S2213-8587(17)30319-4. Epub 2017 Nov 9.
7
Epithelial Na Channel Regulation by Extracellular and Intracellular Factors.上皮钠通道的细胞外和细胞内调节因素。
Annu Rev Physiol. 2018 Feb 10;80:263-281. doi: 10.1146/annurev-physiol-021317-121143. Epub 2017 Nov 9.
8
Dendritic Cell Amiloride-Sensitive Channels Mediate Sodium-Induced Inflammation and Hypertension.树突状细胞阿米洛利敏感通道介导钠诱导的炎症和高血压。
Cell Rep. 2017 Oct 24;21(4):1009-1020. doi: 10.1016/j.celrep.2017.10.002.
9
Amiloride Improves Endothelial Function and Reduces Vascular Stiffness in Female Mice Fed a Western Diet.阿米洛利可改善喂食西式饮食的雌性小鼠的内皮功能并降低血管僵硬度。
Front Physiol. 2017 Jun 30;8:456. doi: 10.3389/fphys.2017.00456. eCollection 2017.
10
Endothelial Mineralocorticoid Receptor Mediates Diet-Induced Aortic Stiffness in Females.内皮盐皮质激素受体介导饮食诱导的雌性主动脉僵硬。
Circ Res. 2016 Mar 18;118(6):935-943. doi: 10.1161/CIRCRESAHA.115.308269. Epub 2016 Feb 15.