参与同基因紫外线诱导肿瘤免疫的淋巴细胞的表型和物理特征。 - Suppr

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超能文献

Phenotypic and physical characteristics of the lymphoid cells involved in the immunity to syngeneic UV-induced tumors.

作者信息

Daynes R A, Schmitt M K, Roberts L K, Spellman C W

出版信息

J Immunol. 1979 Jun;122(6):2458-64.

PMID:312869

Abstract

摘要

相似文献

Phenotypic and physical characteristics of the lymphoid cells involved in the immunity to syngeneic UV-induced tumors.参与同基因紫外线诱导肿瘤免疫的淋巴细胞的表型和物理特征。

J Immunol. 1979 Jun;122(6):2458-64.

Cell-mediated immune response to syngeneic UV-induced tumors. III. Requirement for an Ia+ macrophage in the in vitro differentiation of cytotoxic T lymphocytes.对同基因紫外线诱导肿瘤的细胞介导免疫反应。III. 细胞毒性T淋巴细胞体外分化中Ia+巨噬细胞的需求。

J Immunol. 1979 Apr;122(4):1196-202.

Characterization of a cloned ultraviolet radiation (UV)-induced suppressor T cell line that is capable of inhibiting anti-UV tumor-immune responses.一种能够抑制抗紫外线肿瘤免疫反应的克隆化紫外线诱导抑制性T细胞系的特性分析。

J Immunol. 1986 Mar 1;136(5):1908-16.

Further studies on the tumor-specific suppressor cells induced by ultraviolet radiation.

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Generation of tumor-specific transplantation antigens by UV radiation can occur independently of neoplastic transformation.

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Cell-mediated immune responses to syngeneic tumors. I. Identification of two distinct CTL effector pathways which differ in antigen specificity, genetic regulation, and cell surface phenotype.对同基因肿瘤的细胞介导免疫反应。I. 两种不同CTL效应途径的鉴定，这两种途径在抗原特异性、遗传调控和细胞表面表型方面存在差异。

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Cell-mediated immune response to syngeneic UV-induced tumors. IV. The presence of I-A and I-E subregion-coded antigens on the accessory cell required for the in vitro differentiation of cytotoxic T lymphocytes.

J Immunol. 1979 Sep;123(3):1227-31.

Immune response to progressor variants derived from transfection of an ultraviolet radiation-induced C3H mouse regressor tumor cell line with activated Harvey-ras oncogene.对通过用激活的哈维 - 鼠肉瘤病毒癌基因转染紫外线辐射诱导的C3H小鼠消退肿瘤细胞系所产生的进展变体的免疫反应。

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Immune response to somatic cell hybrids between ultraviolet radiation-induced regressor and spontaneous progressor C3H mouse tumor cells.对紫外线辐射诱导的退化型与自发进展型C3H小鼠肿瘤细胞之间体细胞杂种的免疫反应。

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Cell-mediated immune responses to syngeneic ultraviolet-induced tumours. V. Assessment of accessory and antigen-presenting cell capabilities of normal and ultraviolet-irradiated mice in the generation of anti-tumour cytotoxic effector cells in vitro.对同基因紫外线诱导肿瘤的细胞介导免疫反应。V. 评估正常和紫外线照射小鼠在体外产生抗肿瘤细胞毒性效应细胞过程中的辅助细胞和抗原呈递细胞能力。

Immunology. 1982 Sep;47(1):49-59.

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The contribution of Langerhans cells to cutaneous malignancy.朗格汉斯细胞在皮肤恶性肿瘤中的作用。

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Increase in the ability of human cancer cells to induce cytotoxic T lymphocytes by ultraviolet irradiation.通过紫外线照射提高人类癌细胞诱导细胞毒性T淋巴细胞的能力。

Cancer Immunol Immunother. 1993 Nov;37(6):392-9. doi: 10.1007/BF01526796.

Immunology. 1982 Sep;47(1):49-59.

Effect of ultraviolet radiation on production of epidermal cell thymocyte-activating factor/interleukin 1 in vivo and in vitro.紫外线辐射对体内和体外表皮细胞胸腺细胞激活因子/白细胞介素1产生的影响。

Proc Natl Acad Sci U S A. 1984 Feb;81(4):1198-202. doi: 10.1073/pnas.81.4.1198.

Suppression of contact hypersensitivity by ultraviolet radiation: an experimental model.紫外线辐射对接触性超敏反应的抑制作用：一种实验模型。

Springer Semin Immunopathol. 1981;4(3):293-304. doi: 10.1007/BF01892183.

Ultraviolet light-induced suppression of antigen presentation.紫外线诱导的抗原呈递抑制。

J Clin Immunol. 1983 Apr;3(2):105-10. doi: 10.1007/BF00915480.

Multiple cancers. Tumor burden permits the outgrowth of other cancers.多种癌症。肿瘤负荷会促使其他癌症的发展。

J Exp Med. 1985 Nov 1;162(5):1665-82. doi: 10.1084/jem.162.5.1665.

Identification of a unique tumor antigen as rejection antigen by molecular cloning and gene transfer.通过分子克隆和基因转移鉴定独特的肿瘤抗原作为排斥抗原。

J Exp Med. 1986 Nov 1;164(5):1516-30. doi: 10.1084/jem.164.5.1516.

Antigenic stimulation during ultraviolet therapy in man does not result in immunological tolerance.人体紫外线治疗期间的抗原刺激不会导致免疫耐受。

Clin Exp Immunol. 1989 Apr;76(1):68-72.

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