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C3a and suPAR drive versican V1 expression in tubular cells of focal segmental glomerulosclerosis.

作者信息

Han Runhong, Hu Shuai, Qin Weisong, Shi Jinsong, Hou Qin, Wang Xia, Xu Xiaodong, Zhang Minchao, Zeng Caihong, Liu Zhihong, Bao Hao

出版信息

JCI Insight. 2019 Jul 11;4(13). doi: 10.1172/jci.insight.130986.

DOI:10.1172/jci.insight.130986
PMID:31292294
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC6629242/
Abstract
摘要

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C3a and suPAR drive versican V1 expression in tubular cells of focal segmental glomerulosclerosis.C3a和可溶性尿激酶型纤溶酶原激活物受体（suPAR）驱动局灶节段性肾小球硬化肾小管细胞中多功能蛋白聚糖V1的表达。
JCI Insight. 2019 Jul 11;4(13). doi: 10.1172/jci.insight.130986.
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C3a and suPAR drive versican V1 expression in tubular cells of focal segmental glomerulosclerosis.C3a 和 suPAR 驱动局灶节段性肾小球硬化症中肾小管细胞的 versican V1 表达。
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Serum-soluble urokinase receptor levels do not distinguish focal segmental glomerulosclerosis from other causes of nephrotic syndrome in children.血清可溶性尿激酶受体水平不能区分局灶节段性肾小球硬化与儿童肾病综合征的其他病因。
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Removal of focal segmental glomerulosclerosis (FSGS) factor suPAR using CytoSorb.使用CytoSorb去除局灶节段性肾小球硬化（FSGS）因子可溶性尿激酶型纤溶酶原激活物受体（suPAR）。
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The soluble urokinase receptor is not a clinical marker for focal segmental glomerulosclerosis.可溶性尿激酶型纤溶酶原激活物受体不是局灶节段性肾小球硬化的临床标志物。
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