Retraction: Perinatal Exposure to Bisphenol-A Impairs Spatial Memory through Upregulation of Neurexin1 and Neuroligin3 Expression in Male Mouse Brain.
出版信息
PLoS One. 2019 Jul 18;14(7):e0220212. doi: 10.1371/journal.pone.0220212. eCollection 2019.
Abstract
摘要
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1
Retraction: Perinatal Exposure to Bisphenol-A Impairs Spatial Memory through Upregulation of Neurexin1 and Neuroligin3 Expression in Male Mouse Brain.撤稿声明：围产期暴露于双酚A通过上调雄性小鼠大脑中神经连接蛋白1和神经连接蛋白3的表达损害空间记忆。
PLoS One. 2019 Jul 18;14(7):e0220212. doi: 10.1371/journal.pone.0220212. eCollection 2019.
2
Effect of perinatal exposure to Bisphenol-A on DNA methylation and histone acetylation in cerebral cortex and hippocampus of postnatal male mice.围产期暴露于双酚A对出生后雄性小鼠大脑皮层和海马体中DNA甲基化和组蛋白乙酰化的影响。
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Correction to: Age-related expression of Neurexin1 and Neuroligin3 is correlated with presynaptic density in the cerebral cortex and hippocampus of male mice.对《雄性小鼠大脑皮层和海马中Neurexin1和Neuroligin3的年龄相关表达与突触前密度相关》一文的修正
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Perinatal exposure to low-dose bisphenol A impairs spatial learning and memory in male rats.围产期接触低剂量双酚 A 会损害雄性大鼠的空间学习和记忆能力。
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Perinatal exposure to bisphenol-A impairs learning-memory by concomitant down-regulation of N-methyl-D-aspartate receptors of hippocampus in male offspring mice.围生期暴露于双酚 A 通过同时下调雄性子代小鼠海马 N-甲基-D-天冬氨酸受体损害学习记忆。
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Age-related expression of Neurexin1 and Neuroligin3 is correlated with presynaptic density in the cerebral cortex and hippocampus of male mice.在雄性小鼠的大脑皮层和海马体中，与年龄相关的Neurexin1和Neuroligin3表达与突触前密度相关。
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Adolescent exposure to Bisphenol-A increases anxiety and sucrose preference but impairs spatial memory in rats independent of sex.双酚 A 会增加青少年的焦虑感和蔗糖偏好，但无论性别如何，都会损害其空间记忆。
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Early developmental bisphenol-A exposure sex-independently impairs spatial memory by remodeling hippocampal dendritic architecture and synaptic transmission in rats.早期发育阶段双酚 A 暴露会独立于性别通过重塑大鼠海马树突状结构和突触传递来损害空间记忆。
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Perinatal exposure to genistein, a soy phytoestrogen, improves spatial learning and memory but impairs passive avoidance learning and memory in offspring.围产期暴露于大豆植物雌激素染料木黄酮可改善后代的空间学习和记忆，但会损害其被动回避学习和记忆。
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Anxiety like behavior due to perinatal exposure to Bisphenol-A is associated with decrease in excitatory to inhibitory synaptic density of male mouse brain.围产期暴露于双酚A所导致的焦虑样行为与雄性小鼠大脑兴奋性与抑制性突触密度降低有关。
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本文引用的文献
1
Anxiety like behavior due to perinatal exposure to Bisphenol-A is associated with decrease in excitatory to inhibitory synaptic density of male mouse brain.围产期暴露于双酚A所导致的焦虑样行为与雄性小鼠大脑兴奋性与抑制性突触密度降低有关。
Toxicology. 2017 Mar 1;378:107-113. doi: 10.1016/j.tox.2017.01.010. Epub 2017 Jan 13.
2
Perinatal exposure to bisphenol-A impairs spatial memory through upregulation of neurexin1 and neuroligin3 expression in male mouse brain.围产期暴露于双酚A会通过上调雄性小鼠大脑中神经连接蛋白1和神经配蛋白3的表达来损害空间记忆。
PLoS One. 2014 Oct 17;9(10):e110482. doi: 10.1371/journal.pone.0110482. eCollection 2014.
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