Right on the Nose: IL-17C and Nasal Host Defense.
作者信息
Zemke Anna C, Robinson Keven M
机构信息
Department of MedicineUniversity of Pittsburgh School of MedicinePittsburgh, Pennsylvania.
出版信息
Am J Respir Cell Mol Biol. 2020 Jan;62(1):10-11. doi: 10.1165/rcmb.2019-0236ED.
Abstract
摘要
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本文引用的文献
1
IL-17C Protects Nasal Epithelium from Infection.IL-17C 可保护鼻腔上皮免受感染。
Am J Respir Cell Mol Biol. 2020 Jan;62(1):95-103. doi: 10.1165/rcmb.2018-0377OC.
2
Pseudomonas aeruginosa zinc uptake in chelating environment is primarily mediated by the metallophore pseudopaline.铜绿假单胞菌在螯合环境中的锌摄取主要由金属载体假卟啉介导。
Sci Rep. 2017 Dec 7;7(1):17132. doi: 10.1038/s41598-017-16765-9.
3
Growth of Pseudomonas aeruginosa in zinc poor environments is promoted by a nicotianamine-related metallophore.一种与烟酰胺相关的金属载体可促进铜绿假单胞菌在锌缺乏环境中的生长。
Mol Microbiol. 2017 Nov;106(4):543-561. doi: 10.1111/mmi.13834. Epub 2017 Oct 10.
4
The innate immune protein calprotectin promotes Pseudomonas aeruginosa and Staphylococcus aureus interaction.先天免疫蛋白钙卫蛋白促进铜绿假单胞菌和金黄色葡萄球菌的相互作用。
Nat Commun. 2016 Jun 15;7:11951. doi: 10.1038/ncomms11951.
5
Interleukin-9 and interleukin-17C in chronic rhinosinusitis.慢性鼻-鼻窦炎中的白细胞介素-9和白细胞介素-17C
Int Forum Allergy Rhinol. 2016 Aug;6(8):841-7. doi: 10.1002/alr.21745. Epub 2016 Mar 17.
6
IL-17C expression in nasal epithelial cells of chronic rhinosinusitis with nasal polyposis.白细胞介素-17C 在伴有鼻息肉的慢性鼻-鼻窦炎鼻黏膜上皮细胞中的表达。
Eur Arch Otorhinolaryngol. 2014 May;271(5):1097-105. doi: 10.1007/s00405-013-2683-x. Epub 2013 Sep 7.
7
Toll-like receptor-mediated airway IL-17C enhances epithelial host defense in an autocrine/paracrine manner.Toll 样受体介导的气道 IL-17C 以自分泌/旁分泌方式增强上皮宿主防御。
Am J Respir Cell Mol Biol. 2014 Jan;50(1):30-9. doi: 10.1165/rcmb.2013-0130OC.
8
IL-17C is a mediator of respiratory epithelial innate immune response.IL-17C 是呼吸道上皮固有免疫反应的介质。
Am J Respir Cell Mol Biol. 2013 Apr;48(4):415-21. doi: 10.1165/rcmb.2012-0232OC.
9
Pyoverdine, the Major Siderophore in Pseudomonas aeruginosa, Evades NGAL Recognition.绿脓菌素，铜绿假单胞菌中的主要铁载体，可逃避中性粒细胞明胶酶相关脂质运载蛋白的识别。
Interdiscip Perspect Infect Dis. 2012;2012:843509. doi: 10.1155/2012/843509. Epub 2012 Sep 2.
10
The DeltaF508-CFTR mutation results in increased biofilm formation by Pseudomonas aeruginosa by increasing iron availability.DeltaF508-CFTR突变通过增加铁的可利用性导致铜绿假单胞菌生物膜形成增加。
Am J Physiol Lung Cell Mol Physiol. 2008 Jul;295(1):L25-37. doi: 10.1152/ajplung.00391.2007. Epub 2008 Mar 21.
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