Schulz Christoph U
Ludwig Maximilians University Munich, München, Germany.
J Hand Surg Asian Pac Vol. 2019 Sep;24(3):276-282. doi: 10.1142/S2424835519500346.
Metaphyseal core decompression of the distal radius (MCD) is clinically effective in early lunate necrosis without changing individual wrist mechanics. Its concept is based on the induction of physiologic mechanisms known as physiologic fracture healing response. However, this biological concept does not yet have its place in the historically developed mechanical concepts about Kienböck's disease and requires more detailed clarifications to understand when a change of individual wrist mechanics might be unnecessary. Thirteen consecutive cases, Lichtman stage I (n = 1) or II (n = 12), confirmed by conventional MRI, were treated by MCD. Time off work, changes in magnetic resonance imaging of the lunate, as well as clinical outcome using modified Mayo wrist score were evaluated at final follow-up. Return to work was at six (1-10) weeks after surgery. MRI controls at short-term generally demonstrated stop of progression and signs of bone healing. Independently from ulna variance complete signal normalization was observed in six and a distinct, yet incomplete decrease of lunate bone marrow edema and zones of fat necrosis was confirmed in further six cases after a mean of 21 (13-51) weeks. One patient had radiographic controls only, stating normal healing at 56 months. After a mean follow-up of 37 (12-70) months the clinical outcomes were excellent in eleven and good in two cases (mean 95% in modified Mayo wrist score). In stage I and II lunate necrosis MCD stops disease progression, it improves clinical symptoms and induces normalization of lunate bone signal alterations in MRI. Findings suggest that stage I and II lunate necrosis can be effectively treated without alterations of individual wrist mechanics. Future studies are necessary to readjust common concepts regarding Kienböck's disease, especially focusing on conservative therapy.
桡骨远端干骺端减压术(MCD)在早期月骨坏死的治疗中临床效果显著,且不会改变个体腕关节力学。其理念基于诱导一种被称为生理性骨折愈合反应的生理机制。然而,这一生物学理念在关于月骨无菌性坏死的传统力学理念中尚未占据一席之地,需要更详细的阐释,以便理解何时无需改变个体腕关节力学。连续13例经传统MRI确诊为Lichtman I期(n = 1)或II期(n = 12)的患者接受了MCD治疗。在末次随访时评估其误工时间、月骨磁共振成像的变化以及采用改良Mayo腕关节评分的临床结果。术后6(1 - 10)周恢复工作。短期MRI检查通常显示病情进展停止及骨愈合迹象。无论尺骨变异情况如何,6例患者的月骨信号完全恢复正常,另外6例患者在平均21(13 - 51)周后,月骨骨髓水肿及脂肪坏死区域虽有明显但不完全的减轻。1例患者仅进行了X线检查,显示56个月时愈合正常。平均随访37(12 - 70)个月后,11例患者临床结果为优,2例为良(改良Mayo腕关节评分平均95%)。在I期和II期月骨坏死中,MCD可阻止疾病进展,改善临床症状,并使MRI上月骨信号改变恢复正常。研究结果表明,I期和II期月骨坏死无需改变个体腕关节力学即可得到有效治疗。未来有必要重新调整关于月骨无菌性坏死的常见观念,尤其应关注保守治疗。