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影响食物垃圾通过厌氧消化产生挥发性脂肪酸的因素。

Factors influencing volatile fatty acids production from food wastes via anaerobic digestion.

机构信息

Swedish Centre for Resource Recovery, University of Borås, Borås, Sweden.

Department of Chemical and Polymer Engineering, Faculty of Engineering, Lagos State University, Lagos, Nigeria.

出版信息

Bioengineered. 2020 Dec;11(1):39-52. doi: 10.1080/21655979.2019.1703544.

DOI:10.1080/21655979.2019.1703544
PMID:31880192
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7571609/
Abstract

Volatile fatty acids (VFAs) are intermediate products in anaerobic digestion. The effect of substrate loading or inoculum to substrate ratio (ISR), the addition of methanogen inhibitor, O presence, control the reactor's pH, and inoculum adaptation on the VFAs production from food waste through acidogenesis process was investigated in this study. Addition of 2-bromoethane sulfonic (BES) as methanogen inhibitor suppressed VFA consumption by methanogens at ISR 1:1. At higher substrate loading (ISR 1:3), methane production can be suppressed even without the addition of BES. However, at high substrate loading, controlling the pH during acidogenesis is important to achieve high VFAs yield. Acclimatization of inoculum is also one of the strategies to achieve high VFA yield. The highest VFAs yield obtained in this work was 0.8 g VFA/g VS at ISR 1:3, controlled pH at 6, with the presence of initial O (headspace unflushed).

摘要

挥发性脂肪酸(VFAs)是厌氧消化过程中的中间产物。本研究考察了底物负荷或接种物与底物比(ISR)、添加产甲烷菌抑制剂、O 的存在、控制反应器 pH 值以及接种物适应对通过产酸过程从食物垃圾中生产 VFAs 的影响。添加 2-溴乙烷磺酸(BES)作为产甲烷菌抑制剂可抑制 ISR 为 1:1 时产甲烷菌对 VFA 的消耗。在较高的底物负荷(ISR 1:3)下,即使不添加 BES,也可以抑制甲烷的产生。然而,在高底物负荷下,在产酸过程中控制 pH 值对于获得高 VFAs 产率很重要。接种物的驯化也是获得高 VFA 产率的策略之一。本工作获得的最高 VFAs 产率为 0.8 g VFA/gVS,在 ISR 为 1:3、控制 pH 值为 6、初始 O 存在(未吹扫顶空)的条件下获得。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f58d/7571609/93c1b9fce08a/kbie-11-01-1703544-g009.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f58d/7571609/a1ac2de63657/kbie-11-01-1703544-g002.jpg
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https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f58d/7571609/158f1d61ce03/kbie-11-01-1703544-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f58d/7571609/bb4d6e0c6153/kbie-11-01-1703544-g006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f58d/7571609/b7e046ef8db4/kbie-11-01-1703544-g007.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f58d/7571609/745dec22123d/kbie-11-01-1703544-g008.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f58d/7571609/93c1b9fce08a/kbie-11-01-1703544-g009.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f58d/7571609/a1ac2de63657/kbie-11-01-1703544-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f58d/7571609/e851cf3dceaf/kbie-11-01-1703544-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f58d/7571609/f984db9e5276/kbie-11-01-1703544-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f58d/7571609/158f1d61ce03/kbie-11-01-1703544-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f58d/7571609/bb4d6e0c6153/kbie-11-01-1703544-g006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f58d/7571609/b7e046ef8db4/kbie-11-01-1703544-g007.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f58d/7571609/745dec22123d/kbie-11-01-1703544-g008.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f58d/7571609/93c1b9fce08a/kbie-11-01-1703544-g009.jpg

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