Daniel R H, Hubler W R, Wolf J E, Holder W R
Arch Dermatol. 1977 Jan;113(1):53-6.
Histologic, pharmacologic, and exchange transplant studies were performed on a patient with nevus anemicus. Histologic examination disclosed no abnormalities. The only pharmacologic stimulus that produced erythema within the nevus was the alpha-blocking agent, phentolamine mesylate. Results of the transplant studies demonstrated donor dominance. This suggests that the defect in the nevus anemicus is attributable to increased sensitivity of the blood vessels to catecholamines rather than to increased alpha-adrenergic stimulation.
对一名患有贫血痣的患者进行了组织学、药理学和交换移植研究。组织学检查未发现异常。唯一能使痣内产生红斑的药理学刺激物是α受体阻滞剂甲磺酸酚妥拉明。移植研究结果显示供体占优势。这表明贫血痣的缺陷归因于血管对儿茶酚胺的敏感性增加，而非α肾上腺素能刺激增加。
