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相似文献

1
Erratum: Combining peptide TNIIIA2 with all- retinoic acid accelerates N-Myc protein degradation and neuronal differentiation in MYCN-amplified neuroblastoma cells.勘误:将肽TNIIIA2与全反式维甲酸联合使用可加速MYCN扩增的神经母细胞瘤细胞中N-Myc蛋白的降解和神经元分化。
Am J Cancer Res. 2020 Jan 1;10(1):365-366. eCollection 2020.
2
Combining peptide TNIIIA2 with all- retinoic acid accelerates N-Myc protein degradation and neuronal differentiation in MYCN-amplified neuroblastoma cells.将肽TNIIIA2与全反式维甲酸联合使用可加速MYCN扩增的神经母细胞瘤细胞中N-Myc蛋白的降解和神经元分化。
Am J Cancer Res. 2019 Feb 1;9(2):434-448. eCollection 2019.
3
Acyclic Retinoid Combined With Tenascin-C-derived Peptide Reduces the Malignant Phenotype of Neuroblastoma Cells Through N-Myc Degradation.无环维甲酸联合腱生蛋白-C衍生肽通过降解N-Myc降低神经母细胞瘤细胞的恶性表型。
Anticancer Res. 2019 Jul;39(7):3487-3492. doi: 10.21873/anticanres.13494.
4
A Novel MYCN-Specific Antigene Oligonucleotide Deregulates Mitochondria and Inhibits Tumor Growth in MYCN-Amplified Neuroblastoma.一种新型 MYCN 特异性抗原寡核苷酸可使线粒体失活并抑制 MYCN 扩增神经母细胞瘤的肿瘤生长。
Cancer Res. 2019 Dec 15;79(24):6166-6177. doi: 10.1158/0008-5472.CAN-19-0008. Epub 2019 Oct 15.
5
Vasoactive intestinal peptide decreases MYCN expression and synergizes with retinoic acid in a human MYCN-amplified neuroblastoma cell line.血管活性肠肽可降低MYCN的表达,并在人MYCN扩增的神经母细胞瘤细胞系中与视黄酸协同作用。
Int J Oncol. 2008 Nov;33(5):1081-9.
6
Inhibition of mir-21, which is up-regulated during MYCN knockdown-mediated differentiation, does not prevent differentiation of neuroblastoma cells.在 MYCN 敲低介导的分化过程中上调的 mir-21 的抑制作用并不能阻止神经母细胞瘤细胞的分化。
Differentiation. 2011 Jan;81(1):25-34. doi: 10.1016/j.diff.2010.09.184. Epub 2010 Oct 25.
7
Positive auto-regulation of MYCN in human neuroblastoma.MYCN在人类神经母细胞瘤中的正向自我调节。
Biochem Biophys Res Commun. 2009 Dec 4;390(1):21-6. doi: 10.1016/j.bbrc.2009.09.044. Epub 2009 Sep 18.
8
p19-INK4d inhibits neuroblastoma cell growth, induces differentiation and is hypermethylated and downregulated in MYCN-amplified neuroblastomas.p19-INK4d抑制神经母细胞瘤细胞生长,诱导分化,并且在MYCN扩增的神经母细胞瘤中发生高甲基化并下调。
Hum Mol Genet. 2014 Dec 20;23(25):6826-37. doi: 10.1093/hmg/ddu406. Epub 2014 Aug 7.
9
Distinct transcriptional MYCN/c-MYC activities are associated with spontaneous regression or malignant progression in neuroblastomas.MYCN/c-MYC 的转录活性差异与神经母细胞瘤的自发消退或恶性进展相关。
Genome Biol. 2008 Oct 13;9(10):R150. doi: 10.1186/gb-2008-9-10-r150.
10
-amplified neuroblastoma maintains an aggressive and undifferentiated phenotype by deregulation of estrogen and NGF signaling.-扩增的神经母细胞瘤通过雌激素和 NGF 信号的失调来维持侵袭性和未分化的表型。
Proc Natl Acad Sci U S A. 2018 Feb 6;115(6):E1229-E1238. doi: 10.1073/pnas.1710901115. Epub 2018 Jan 26.

勘误:将肽TNIIIA2与全反式维甲酸联合使用可加速MYCN扩增的神经母细胞瘤细胞中N-Myc蛋白的降解和神经元分化。

Erratum: Combining peptide TNIIIA2 with all- retinoic acid accelerates N-Myc protein degradation and neuronal differentiation in MYCN-amplified neuroblastoma cells.

作者信息

Otsuka Kazuki, Sasada Manabu, Iyoda Takuya, Nohara Yusuke, Sakai Shunsuke, Asayama Tatsufumi, Suenaga Yusuke, Yokoi Sana, Higami Yoshikazu, Kodama Hiroaki, Fukai Fumio

机构信息

Department of Molecular Patho-Physiology, Faculty of Pharmaceutical Sciences, Tokyo University of Science Noda, Chiba, Japan.

Translational Research Center, Research Institutes for Science and Technology, Tokyo University of Science Noda, Chiba, Japan.

出版信息

Am J Cancer Res. 2020 Jan 1;10(1):365-366. eCollection 2020.

PMID:32064173
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7017727/
Abstract

[This corrects the article on p. 434 in vol. 9, PMID: 30906641.].

摘要

[这更正了第9卷第434页的文章,PMID: 30906641。]