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1
Cardiac Neural Crest.心脏神经嵴。
Cold Spring Harb Perspect Biol. 2021 Jan 4;13(1):a036715. doi: 10.1101/cshperspect.a036715.
2
Cardiac outflow tract anomalies.心脏流出道异常。
Wiley Interdiscip Rev Dev Biol. 2013 Jul;2(4):499-530. doi: 10.1002/wdev.98. Epub 2013 Feb 19.
3
HAND transcription factors cooperatively specify the aorta and pulmonary trunk.HAND 转录因子协同指定主动脉和肺动脉。
Dev Biol. 2021 Aug;476:1-10. doi: 10.1016/j.ydbio.2021.03.011. Epub 2021 Mar 20.
4
Cardiac outflow tract defects in mice lacking ALK2 in neural crest cells.神经嵴细胞中缺乏ALK2的小鼠的心脏流出道缺陷。
Development. 2004 Jul;131(14):3481-90. doi: 10.1242/dev.01214.
5
Rac1 Signaling Is Required for Anterior Second Heart Field Cellular Organization and Cardiac Outflow Tract Development.Rac1信号传导对于前侧第二心脏场细胞组织和心脏流出道发育是必需的。
J Am Heart Assoc. 2015 Dec 31;5(1):e002508. doi: 10.1161/JAHA.115.002508.
6
Cardiac neural crest contribution to the pulmonary artery and sixth aortic arch artery complex in chick embryos aged 6 to 18 days.心脏神经嵴对6至18日龄鸡胚肺动脉和第六主动脉弓动脉复合体的贡献。
Anat Rec. 1993 Nov;237(3):385-99. doi: 10.1002/ar.1092370312.
7
Hemodynamic changes in chick embryos precede heart defects after cardiac neural crest ablation.心脏神经嵴消融后，鸡胚的血流动力学变化先于心脏缺陷出现。
Circ Res. 1986 Nov;59(5):545-50. doi: 10.1161/01.res.59.5.545.
8
Temporospatial study of the migration and distribution of cardiac neural crest in quail-chick chimeras.鹌鹑-鸡嵌合体中心脏神经嵴迁移与分布的时空研究。
Am J Anat. 1991 Sep;192(1):79-88. doi: 10.1002/aja.1001920109.
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Histone deacetylase 3 regulates smooth muscle differentiation in neural crest cells and development of the cardiac outflow tract.组蛋白去乙酰化酶 3 调控神经嵴细胞中平滑肌的分化和心流出道的发育。
Circ Res. 2011 Nov 11;109(11):1240-9. doi: 10.1161/CIRCRESAHA.111.255067. Epub 2011 Sep 29.
10
Distinct cardiac malformations caused by absence of connexin 43 in the neural crest and in the non-crest neural tube.神经嵴和非嵴神经管中连接蛋白43缺失导致的不同心脏畸形。
Development. 2006 May;133(10):2063-73. doi: 10.1242/dev.02374. Epub 2006 Apr 19.
引用本文的文献
1
Cardiovascular Mettl3 Deficiency Causes Congenital Cardiac Defects and Postnatal Lethality in Mice.心血管系统中Mettl3缺乏导致小鼠先天性心脏缺陷和出生后死亡。
Int J Biol Sci. 2025 Mar 10;21(6):2430-2445. doi: 10.7150/ijbs.100941. eCollection 2025.
2
The avian embryo as a time-honoured animal model in developmental, biomedical and agricultural research.鸟类胚胎作为发育、生物医学和农业研究中历史悠久的动物模型。
Philos Trans R Soc Lond B Biol Sci. 2025 Feb 27;380(1920):20230438. doi: 10.1098/rstb.2023.0438.
3
Gene expression patterns of the developing human face at single cell resolution reveal cell type contributions to normal facial variation and disease risk.单细胞分辨率下发育中人类面部的基因表达模式揭示了细胞类型对正常面部变异和疾病风险的影响。
bioRxiv. 2025 Feb 5:2025.01.18.633396. doi: 10.1101/2025.01.18.633396.
4
Polarity and migration of cranial and cardiac neural crest cells: underlying molecular mechanisms and disease implications.颅面和心脏神经嵴细胞的极性与迁移：潜在分子机制及疾病影响
Front Cell Dev Biol. 2025 Jan 6;12:1457506. doi: 10.3389/fcell.2024.1457506. eCollection 2024.
5
Timing and Graded BMP Signalling Determines Fate of Neural Crest and Ectodermal Placode Derivatives from Pluripotent Stem Cells.时间和分级BMP信号决定多能干细胞来源的神经嵴和外胚层基板衍生物的命运。
Biomedicines. 2024 Oct 4;12(10):2262. doi: 10.3390/biomedicines12102262.
6
[Maternal gene polymorphisms and their interactions with periconceptional folic acid supplementation in relation to offspring ventricular septal defects].[母亲基因多态性及其与孕期叶酸补充剂的相互作用与子代室间隔缺损的关系]
Zhongguo Dang Dai Er Ke Za Zhi. 2024;26(9):899-906. doi: 10.7499/j.issn.1008-8830.2403067.
7
The chromatin regulator Ankrd11 controls cardiac neural crest cell-mediated outflow tract remodeling and heart function.染色质调控因子 Ankrd11 控制心脏神经嵴细胞介导的流出道重塑和心脏功能。
Nat Commun. 2024 Jul 1;15(1):4632. doi: 10.1038/s41467-024-48955-1.
8
Electrocardiographic abnormalities in patients with microtia.小耳畸形患者的心电图异常
Sci Rep. 2024 May 3;14(1):10191. doi: 10.1038/s41598-024-60610-9.
9
Systematic review of cardiovascular neurocristopathy-contemporary insights and future perspectives.心血管神经嵴病的系统评价——当代见解与未来展望
Front Cardiovasc Med. 2024 Apr 9;11:1333265. doi: 10.3389/fcvm.2024.1333265. eCollection 2024.
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Whole exome sequencing reveals genetic landscape associated with left ventricular outflow tract obstruction in Chinese Han population.全外显子组测序揭示了中国汉族人群中与左心室流出道梗阻相关的遗传图谱。
Front Genet. 2023 Dec 18;14:1267368. doi: 10.3389/fgene.2023.1267368. eCollection 2023.
本文引用的文献
1
Regulation of Sema3c and the Interaction between Cardiac Neural Crest and Second Heart Field during Outflow Tract Development.Sema3c 的调控及心神经嵴与第二心区在流出道发育中的相互作用。
Sci Rep. 2017 Jul 28;7(1):6771. doi: 10.1038/s41598-017-06964-9.
2
Heparan Sulfate Biosynthesis Enzyme, Ext1, Contributes to Outflow Tract Development of Mouse Heart via Modulation of FGF Signaling.硫酸乙酰肝素生物合成酶Ext1通过调节FGF信号通路促进小鼠心脏流出道发育。
PLoS One. 2015 Aug 21;10(8):e0136518. doi: 10.1371/journal.pone.0136518. eCollection 2015.
3
A critical role for the chromatin remodeller CHD7 in anterior mesoderm during cardiovascular development.染色质重塑因子CHD7在心血管发育过程中前中胚层发挥关键作用。
Dev Biol. 2015 Sep 1;405(1):82-95. doi: 10.1016/j.ydbio.2015.06.017. Epub 2015 Jun 21.
4
Neural crest-derived SEMA3C activates endothelial NRP1 for cardiac outflow tract septation.神经嵴衍生的SEMA3C激活内皮细胞的NRP1以实现心脏流出道分隔。
J Clin Invest. 2015 Jul 1;125(7):2661-76. doi: 10.1172/JCI79668. Epub 2015 Jun 8.
5
α5 and αv integrins cooperate to regulate vascular smooth muscle and neural crest functions in vivo.α5和αv整合素在体内协同调节血管平滑肌和神经嵴功能。
Development. 2015 Feb 15;142(4):797-808. doi: 10.1242/dev.117572.
6
Inappropriate p53 activation during development induces features of CHARGE syndrome.发育过程中不当的 p53 激活可诱导 CHARGE 综合征的特征。
Nature. 2014 Oct 9;514(7521):228-32. doi: 10.1038/nature13585. Epub 2014 Aug 3.
7
Endothelial cells regulate neural crest and second heart field morphogenesis.内皮细胞调节神经嵴和第二心区形态发生。
Biol Open. 2014 Jul 4;3(8):679-88. doi: 10.1242/bio.20148078.
8
CHD7, the gene mutated in CHARGE syndrome, regulates genes involved in neural crest cell guidance.CHD7基因在CHARGE综合征中发生突变，它调控参与神经嵴细胞导向的基因。
Hum Genet. 2014 Aug;133(8):997-1009. doi: 10.1007/s00439-014-1444-2. Epub 2014 Apr 13.
9
Requirement for integrin-linked kinase in neural crest migration and differentiation and outflow tract morphogenesis.整联蛋白连接激酶在神经嵴迁移、分化和流出道形态发生中的作用。
BMC Biol. 2013 Oct 16;11:107. doi: 10.1186/1741-7007-11-107.
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Neural crest cells are required for correct positioning of the developing outflow cushions and pattern the arterial valve leaflets.神经嵴细胞对于流出道垫的正确定位和动脉瓣叶的形态发生是必需的。
Cardiovasc Res. 2013 Aug 1;99(3):452-60. doi: 10.1093/cvr/cvt132. Epub 2013 May 30.
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