Yamashita T, Kozuki K, Kubo N, Ishibe T, Kumazawa H, Otani K, Kumazawa T
Department of Otolaryngology, Kansai Medical University, Osaka, Japan.
Acta Otolaryngol Suppl. 1988;454:237-40. doi: 10.3109/00016488809125034.
Participation of the autonomic nerve in tonsillar focal infection was investigated by measuring neurotransmitters, receptors and microvibration. In focal infection patients, the volume of norepinephrine in the tonsil increased significantly and the number of a-adrenergic receptors decreased. These findings suggest that the focally infected tonsil exhibits a high degree of sympathetic nerve activity. The ratio of N-type in microvibration decreased systematically, which indicates some imbalance or immaturity of the autonomic nervous system in focal infection patients. Based on this data, the production mechanism of tonsillar focal infection was speculated from the point of view of the autonomic nerve.
通过测量神经递质、受体和微振动来研究自主神经在扁桃体局灶性感染中的参与情况。在局灶性感染患者中，扁桃体中去甲肾上腺素的含量显著增加，α-肾上腺素能受体的数量减少。这些发现表明，局灶性感染的扁桃体表现出高度的交感神经活动。微振动中N型的比例系统性降低，这表明局灶性感染患者的自主神经系统存在某种失衡或不成熟。基于这些数据，从自主神经的角度推测了扁桃体局灶性感染的产生机制。
