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来那度胺联合全反式维甲酸治疗不适合强化治疗的复发/难治性 AML 患者中 LSD1 抑制曲安西龙的概念验证 I/II 期试验

A proof of concept phase I/II pilot trial of LSD1 inhibition by tranylcypromine combined with ATRA in refractory/relapsed AML patients not eligible for intensive therapy.

机构信息

Department of Hematology and Oncology, University Hospital Halle, Halle (Saale), Germany.

Department of Medicine V, Hematology, Oncology and Rheumatology, University Hospital Heidelberg, Heidelberg, Germany.

出版信息

Leukemia. 2021 Mar;35(3):701-711. doi: 10.1038/s41375-020-0892-z. Epub 2020 Jun 19.

DOI:10.1038/s41375-020-0892-z
PMID:32561840
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7303943/
Abstract

All-trans-retinoic acid (ATRA) is highly active in acute promyelocytic leukemia but not in other types of acute myeloid leukemia (AML). Previously, we showed that ATRA in combination with Lysine-specific demethylase 1 (LSD1) inhibition by tranylcypromine (TCP) can induce myeloid differentiation in AML blasts. This phase I/II clinical trial investigated the safety and efficacy of TCP/ATRA treatment as salvage therapy for relapsed/refractory (r/r) AML. The combination was evaluated in 18 patients, ineligible for intensive treatment. The overall response rate was 20%, including two complete remissions without hematological recovery and one partial response. We also observed myeloid differentiation upon TCP/ATRA treatment in patients who did not reach clinical remission. Median overall survival (OS) was 3.3 months, and one-year OS 22%. One patient developed an ATRA-induced differentiation syndrome. The most frequently reported adverse events were vertigo and hypotension. TCP plasma levels correlated with intracellular TCP concentration. Increased H3K4me1 and H3k4me2 levels were observed in AML blasts and white blood cells from some TCP/ATRA treated patients. Combined TCP/ATRA treatment can induce differentiation of AML blasts and lead to clinical response in heavily pretreated patients with r/r AML with acceptable toxicity. These findings emphasize the potential of LSD1 inhibition combined with ATRA for AML treatment.

摘要

全反式维甲酸(ATRA)在急性早幼粒细胞白血病中高度活跃,但在其他类型的急性髓系白血病(AML)中则不然。此前,我们发现 ATRA 与赖氨酸特异性去甲基化酶 1(LSD1)抑制剂曲普利啶(TCP)联合使用可以诱导 AML 细胞分化。这项 I/II 期临床试验研究了 TCP/ATRA 作为复发/难治性(r/r)AML 挽救治疗的安全性和疗效。该联合治疗方案评估了 18 名不符合强化治疗条件的患者。总体缓解率为 20%,包括 2 例无血液学恢复的完全缓解和 1 例部分缓解。我们还观察到在未达到临床缓解的患者中,TCP/ATRA 治疗后出现了髓系分化。中位总生存期(OS)为 3.3 个月,1 年 OS 为 22%。1 名患者出现 ATRA 诱导的分化综合征。最常报告的不良反应是眩晕和低血压。TCP 血浆水平与细胞内 TCP 浓度相关。在 AML 细胞和一些接受 TCP/ATRA 治疗的患者的白细胞中观察到 H3K4me1 和 H3k4me2 水平增加。联合 TCP/ATRA 治疗可诱导 AML 细胞分化,并在接受过多重预处理的 r/r AML 患者中引起临床反应,具有可接受的毒性。这些发现强调了 LSD1 抑制联合 ATRA 治疗 AML 的潜力。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/26f7/7303943/5ee174b7d520/41375_2020_892_Fig6_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/26f7/7303943/d904e400523b/41375_2020_892_Fig1_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/26f7/7303943/7029f70617a9/41375_2020_892_Fig2_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/26f7/7303943/4538446737c0/41375_2020_892_Fig3_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/26f7/7303943/9f7fa0406905/41375_2020_892_Fig4_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/26f7/7303943/a7d0e0adae55/41375_2020_892_Fig5_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/26f7/7303943/5ee174b7d520/41375_2020_892_Fig6_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/26f7/7303943/d904e400523b/41375_2020_892_Fig1_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/26f7/7303943/7029f70617a9/41375_2020_892_Fig2_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/26f7/7303943/4538446737c0/41375_2020_892_Fig3_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/26f7/7303943/9f7fa0406905/41375_2020_892_Fig4_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/26f7/7303943/a7d0e0adae55/41375_2020_892_Fig5_HTML.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/26f7/7303943/5ee174b7d520/41375_2020_892_Fig6_HTML.jpg

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