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本文引用的文献
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Interpersonal Gut Microbiome Variation Drives Susceptibility and Resistance to Cholera Infection.
Cell. 2020 Jun 25;181(7):1533-1546.e13. doi: 10.1016/j.cell.2020.05.036. Epub 2020 Jun 16.
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Gut microbiome communication with bone marrow regulates susceptibility to amebiasis.
J Clin Invest. 2020 Aug 3;130(8):4019-4024. doi: 10.1172/JCI133605.
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Strain-Dependent Inhibition of Clostridioides difficile by Commensal Carrying the Bile Acid-Inducible () Operon.
J Bacteriol. 2020 May 11;202(11). doi: 10.1128/JB.00039-20.
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Bile acid metabolites control T17 and T cell differentiation.
Nature. 2019 Dec;576(7785):143-148. doi: 10.1038/s41586-019-1785-z. Epub 2019 Nov 27.
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Diversification of host bile acids by members of the gut microbiota.
Gut Microbes. 2020;11(2):158-171. doi: 10.1080/19490976.2019.1674124. Epub 2019 Oct 9.
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Bile salt hydrolases: Gatekeepers of bile acid metabolism and host-microbiome crosstalk in the gastrointestinal tract.
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Restoration of short chain fatty acid and bile acid metabolism following fecal microbiota transplantation in patients with recurrent Clostridium difficile infection.
Anaerobe. 2018 Oct;53:64-73. doi: 10.1016/j.anaerobe.2018.04.001. Epub 2018 Apr 12.
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Recurrent Clostridium difficile infection associates with distinct bile acid and microbiome profiles.
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Updated global burden of cholera in endemic countries.
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