使用15-甲基前列腺素F2α治疗宫缩乏力导致的母体动脉血氧饱和度降低。
Maternal arterial desaturation with 15-methyl prostaglandin F2 alpha for uterine atony.
作者信息
Hankins G D, Berryman G K, Scott R T, Hood D
机构信息
Department of Obstetrics and Gynecology, Wilford Hall United States Air Force Medical Center, Lackland Air Force Base, Texas.
出版信息
Obstet Gynecol. 1988 Sep;72(3 Pt 1):367-70.
Over a six-month period, five women with severe uterine atony and postpartum hemorrhage developed marked maternal arterial oxygen desaturation within five to ten minutes of the administration of 15-methyl prostaglandin F2 alpha. The average fall from baseline was 10.4 +/- 5.4%, to a mean arterial oxygen saturation of 88.8 +/- 5.45%. The desaturation was accompanied by acute increases, averaging 20.7 +/- 5.9%, in the intrapulmonary shunt.
在六个月的时间里，五名患有严重子宫收缩乏力和产后出血的女性在注射15-甲基前列腺素F2α后五到十分钟内出现明显的母体动脉血氧饱和度降低。与基线相比平均下降了10.4±5.4%，平均动脉血氧饱和度降至88.8±5.45%。这种血氧饱和度降低伴随着肺内分流平均急性增加20.7±5.9%。
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