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Corrigendum to "OCT3/4-binding sequence-dependent maintenance of the unmethylated state of CTCF-binding sequences with DNA demethylation and suppression of de novo DNA methylation in the H19 imprinted control region" [Gene 743 (2020) 144606].

作者信息

Hori Naohiro, Kubo Shuichi, Sakasegawa Taku, Sakurai Chiye, Hatsuzawa Kiyotaka

机构信息

Division of Molecular Biology, Department of Molecular and Cellular Biology, School of Life Sciences, Faculty of Medicine, Tottori University, Nishi-cho 86, Yonago, Tottori, Japan.

Division of Molecular Biology, Department of Molecular and Cellular Biology, School of Life Sciences, Faculty of Medicine, Tottori University, Nishi-cho 86, Yonago, Tottori, Japan.

出版信息

Gene. 2021 Feb 15;769:144923. doi: 10.1016/j.gene.2020.144923. Epub 2020 Jul 1.

DOI:10.1016/j.gene.2020.144923
PMID:32622804
Abstract
摘要

相似文献

1
Corrigendum to "OCT3/4-binding sequence-dependent maintenance of the unmethylated state of CTCF-binding sequences with DNA demethylation and suppression of de novo DNA methylation in the H19 imprinted control region" [Gene 743 (2020) 144606].对“OCT3/4结合序列依赖性维持CTCF结合序列的未甲基化状态并伴有DNA去甲基化以及抑制H19印记控制区域中的从头DNA甲基化”[《基因》743 (2020) 144606]的勘误
Gene. 2021 Feb 15;769:144923. doi: 10.1016/j.gene.2020.144923. Epub 2020 Jul 1.
2
OCT3/4-binding sequence-dependent maintenance of the unmethylated state of CTCF-binding sequences with DNA demethylation and suppression of de novo DNA methylation in the H19 imprinted control region.OCT3/4 结合序列依赖性地维持 CTCF 结合序列的非甲基化状态，同时在 H19 印迹控制区抑制从头 DNA 甲基化。
Gene. 2020 Jun 15;743:144606. doi: 10.1016/j.gene.2020.144606. Epub 2020 Mar 19.
3
Changes in the DNA methylation profile of the rat H19 gene upstream region during development and transgenic hepatocarcinogenesis and its role in the imprinted transcriptional regulation of the H19 gene.大鼠H19基因上游区域在发育和转基因肝癌发生过程中的DNA甲基化谱变化及其在H19基因印记转录调控中的作用。
Mol Carcinog. 2004 Sep;41(1):1-16. doi: 10.1002/mc.20036.
4
Sox-Oct motifs contribute to maintenance of the unmethylated H19 ICR in YAC transgenic mice.Sox-Oct 基序有助于维持 YAC 转基因小鼠中未甲基化的 H19 ICR。
Hum Mol Genet. 2013 Nov 15;22(22):4627-37. doi: 10.1093/hmg/ddt311. Epub 2013 Jul 2.
5
CTCF mediates methylation-sensitive enhancer-blocking activity at the H19/Igf2 locus.CCCTC结合因子（CTCF）介导H19/胰岛素样生长因子2（Igf2）基因座处的甲基化敏感增强子阻断活性。
Nature. 2000 May 25;405(6785):486-9. doi: 10.1038/35013106.
6
CTCF binding is not the epigenetic mark that establishes post-fertilization methylation imprinting in the transgenic H19 ICR.CTCF 结合并不是在转基因 H19 ICR 中建立受精后甲基化印迹的表观遗传标记。
Hum Mol Genet. 2010 Apr 1;19(7):1190-8. doi: 10.1093/hmg/ddp589. Epub 2010 Jan 4.
7
CTCF elements direct allele-specific undermethylation at the imprinted H19 locus.CTCF元件指导印记H19基因座的等位基因特异性低甲基化。
Curr Biol. 2004 Jun 8;14(11):1007-12. doi: 10.1016/j.cub.2004.05.041.
8
Synthetic DNA fragments bearing ICR cis elements become differentially methylated and recapitulate genomic imprinting in transgenic mice.带有 ICR 顺式元件的合成 DNA 片段会发生差异甲基化，并在转基因小鼠中重现基因组印记。
Epigenetics Chromatin. 2018 Jun 29;11(1):36. doi: 10.1186/s13072-018-0207-z.
9
CTCF binding at the insulin-like growth factor-II (IGF2)/H19 imprinting control region is insufficient to regulate IGF2/H19 expression in human tissues.CTCF在胰岛素样生长因子-II（IGF2）/H19印记控制区域的结合不足以调节人类组织中IGF2/H19的表达。
Endocrinology. 2003 Oct;144(10):4420-6. doi: 10.1210/en.2003-0681. Epub 2003 Jul 10.
10
De novo DNA methylation through the 5'-segment of the H19 ICR maintains its imprint during early embryogenesis.通过H19基因印记控制区5'-片段的从头DNA甲基化在早期胚胎发育过程中维持其印记。
Development. 2015 Nov 15;142(22):3833-44. doi: 10.1242/dev.126003. Epub 2015 Sep 28.

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