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Erratum. Metformin inhibits testosterone-induced endoplasmic reticulum stress in ovarian granulosa cells via inactivation of p38 MAPK.勘误。二甲双胍通过使p38丝裂原活化蛋白激酶失活来抑制睾酮诱导的卵巢颗粒细胞内质网应激。
Hum Reprod. 2020 Aug 1;35(8):1947-1948. doi: 10.1093/humrep/deaa180.
2
Endoplasmic Reticulum Stress Activated by Androgen Enhances Apoptosis of Granulosa Cells via Induction of Death Receptor 5 in PCOS.雄激素激活内质网应激通过诱导多囊卵巢综合征中死亡受体 5 增强颗粒细胞凋亡。
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Low glucose and metformin-induced apoptosis of human ovarian cancer cells is connected to ASK1 via mitochondrial and endoplasmic reticulum stress-associated pathways.低糖和二甲双胍诱导的人卵巢癌细胞凋亡与 ASK1 通过线粒体和内质网应激相关途径有关。
J Exp Clin Cancer Res. 2019 Feb 13;38(1):77. doi: 10.1186/s13046-019-1090-6.
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Metformin potentiates the effect of arsenic trioxide suppressing intrahepatic cholangiocarcinoma: roles of p38 MAPK, ERK3, and mTORC1.二甲双胍增强三氧化二砷抑制肝内胆管癌的作用：p38丝裂原活化蛋白激酶、ERK3和雷帕霉素靶蛋白复合物1的作用
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Role of p38 mitogen-activated protein kinase in linking stearoyl-CoA desaturase-1 activity with endoplasmic reticulum homeostasis.p38丝裂原活化蛋白激酶在将硬脂酰辅酶A去饱和酶-1活性与内质网稳态联系起来中的作用。
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Alleviation of endoplasmic reticulum stress protects against cisplatin-induced ovarian damage.减轻内质网应激可预防顺铂诱导的卵巢损伤。
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Suppressive effects of FR167653, an inhibitor of p38 mitogen-activated kinase, on calreticulin mRNA expression induced by endoplasmic reticulum stresses.p38丝裂原活化蛋白激酶抑制剂FR167653对内质网应激诱导的钙网蛋白mRNA表达的抑制作用
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Angiotensin II-induced podocyte apoptosis is mediated by endoplasmic reticulum stress/PKC-δ/p38 MAPK pathway activation and trough increased Na/H exchanger isoform 1 activity.血管紧张素II诱导的足细胞凋亡是由内质网应激/PKC-δ/p38丝裂原活化蛋白激酶途径激活介导的，并通过增加钠/氢交换体1的活性来实现。
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[P38 MAPK signaling pathway mediates advanced oxidation protein product-induced epithelial-to-mesenchymal transition in tubular cells].[P38丝裂原活化蛋白激酶信号通路介导晚期氧化蛋白产物诱导的肾小管上皮细胞向间充质细胞转化]
Nan Fang Yi Ke Da Xue Xue Bao. 2016 Aug 20;36(9):1209-1214.
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Androgens Increase Accumulation of Advanced Glycation End Products in Granulosa Cells by Activating ER Stress in PCOS.雄激素通过激活 PCOS 中内质网应激增加颗粒细胞中晚期糖基化终产物的积累。
Endocrinology. 2020 Feb 1;161(2). doi: 10.1210/endocr/bqaa015.

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Metformin: A promising clinical therapeutical approach for BPH treatment via inhibiting dysregulated steroid hormones-induced prostatic epithelial cells proliferation.二甲双胍：一种通过抑制失调的类固醇激素诱导的前列腺上皮细胞增殖来治疗良性前列腺增生的有前景的临床治疗方法。
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cFLIP Alleviates Myocardial Ischemia-Reperfusion Injury by Inhibiting Endoplasmic Reticulum Stress.cFLIP通过抑制内质网应激减轻心肌缺血再灌注损伤。
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Erratum. Metformin inhibits testosterone-induced endoplasmic reticulum stress in ovarian granulosa cells via inactivation of p38 MAPK.

作者信息

Jin Jiamin, Ma Yerong, Tong Xiaomei, Yang Weijie, Dai Yongdong, Pan Yibin, Ren Peipei, Liu Liu, Fan Heng-Yu, Zhang Yinli, Zhang Songying

机构信息

Assisted Reproduction Unit, Department of Obstetrics and Gynecology, Sir Run Run Shaw Hospital, Zhejiang University School of Medicine, 310016, Hangzhou, China.

Department of Obstetrics and Gynecology, Key Laboratory of Reproductive Dysfunction Management of Zhejiang Province, 310016, Hangzhou, China.

出版信息

Hum Reprod. 2020 Aug 1;35(8):1947-1948. doi: 10.1093/humrep/deaa180.

DOI:10.1093/humrep/deaa180
PMID:32644121
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7398621/
Abstract
摘要