Grimes W R., Stratton Michael
LSU School of Medicine Shreveport
LSU Shreveport Health Sciences Center
Pelvic floor dysfunction (PFD) refers to a broad constellation of symptoms and anatomic changes related to abnormal function of the pelvic floor musculature. The disordered function corresponds to either increase activity (hypertonicity) or diminished activity (hypotonicity) or inappropriate coordination of the pelvic floor muscles. Alterations regarding the support of pelvic organs are included in the discussion of PFD and are known as Pelvic Organ Prolapse (POP). The clinical aspects of PFD can be urologic, gynecologic, or colorectal and are often interrelated. Another way to compartmentalize the concerns are anterior- urethra/bladder, middle- vagina/uterus and posterior- anus/rectum. The pelvic floor is a combination of multiple muscles with ligamentous attachments creating a dome-shaped diaphragm across the boney pelvic outlet. This complex of muscles spans from the pubis (anterior) to the sacrum/coccyx (posterior) and bilateral to the ischial tuberosities. The bulk of the pelvic musculature is the levator ani, composed of the puborectalis, pubococcygeus, and iliococcygeus. The puborectalis wraps as a sling around the anorectal junction accentuating the anorectal angle during contraction and is a primary contributor to fecal continence. Elevation and support of the pelvic organs are associated with the pubococcygeus and the iliococcygeus. The pubococcygeus is the most medial component which separates, fashioning the levator hiatus with openings for the urethra, vagina (females), and anus. The bulbospongiosus and ischiocavernosus muscles are the primary contributors to the superficial portion of the anterior pelvic floor. The more superficial musculature of the posterior pelvic floor constitutes the external anal sphincter. The transverse perineal muscles cross the mid-portion of the superficial aspect of the pelvic floor and coalesce with the bulbospongiosus muscles and external anal sphincter as the perineal body. The nerve supply to the pelvic floor structures is primarily from sacral nerves S3 and S4 as the pudendal nerve. The predominant blood supply is derived from parietal branches of the internal iliac artery.The muscles of the pelvic floor have three functions: 1. Support of the pelvic organs- bladder, urethra, prostate (males), vagina and uterus (females), anus, and rectum, along with the general support of the intra-abdominal contents. 2. Contribute to continence of urine and feces. 3. Contribute to the sexual functions of arousal and orgasm. A wide variety of conditions are attributed to PFD due to hypertonicity, hypotonicity, loss of pelvic support, or mixed concerns. Urologic: Difficult urination: hesitancy, delay in the urinary stream. Cystocele: bulging or herniation of the bladder into the vagina (anterior). Urethrocele(urethral prolapse): bulging of the urethra into the vagina (anterior). Urinary incontinence: involuntary leakage of urine. Gynecologic: Dyspareunia: pain with or following sexual intercourse. Uterine prolapse: herniation of the uterus via the vagina beyond the introitus. Vaginal prolapse: herniation of the vaginal apex beyond the introitus. Enterocele: bulging or herniation of the intestines into the vagina (apical/posterior). Rectocele: bulging or herniation of the rectum into the vagina (posterior). Colorectal: Constipation: paradoxical contraction or inadequate relaxation of the pelvic floor muscles during attempted defecation (dyssynergic defecation). Fecal incontinence: involuntary leakage of stool (not related to sphincter disruption). Rectal prolapse: intussusception of the rectum beyond the anal verge (Procedentia) or proximal to the anus (Occult). General: Pelvic pain: chronic pain lasting more than three to six months, unrelated to other defined conditions. Levator spasm: another term for chronic pelvic pain related to the levator ani musculature. Proctalgia fugax: fleeting spastic pain related to the levator ani musculature. Perineal descent- bulging of the perineum below the boney pelvic outlet.
盆底功能障碍(PFD)指的是一系列与盆底肌肉组织功能异常相关的症状和解剖学变化。功能紊乱表现为盆底肌肉活动增加(张力亢进)、活动减少(张力减退)或协调性异常。盆底功能障碍的讨论范畴包括盆腔器官支撑结构的改变,即盆腔器官脱垂(POP)。PFD的临床症状可涉及泌尿系统、妇科或结直肠领域,且常常相互关联。另一种分类方式是分为前部(尿道/膀胱)、中部(阴道/子宫)和后部(肛门/直肠)。盆底是由多块肌肉与韧带附着组合而成,在骨盆出口处形成一个穹顶状的膈膜。这组肌肉从耻骨(前部)延伸至骶骨/尾骨(后部),并向两侧延伸至坐骨结节。盆底大部分肌肉是肛提肌,由耻骨直肠肌、耻骨尾骨肌和髂尾骨肌组成。耻骨直肠肌像吊索一样环绕在肛管直肠交界处,收缩时使肛管直肠角增大,是粪便失禁的主要影响因素。盆腔器官的提升和支撑与耻骨尾骨肌和髂尾骨肌有关。耻骨尾骨肌是最内侧的部分,将其分开,形成提肌裂孔,有尿道、阴道(女性)和肛门的开口。球海绵体肌和坐骨海绵体肌是盆底前部浅表部分的主要组成部分。盆底后部较浅表的肌肉构成肛门外括约肌。会阴横肌穿过盆底浅表部分的中部,并与球海绵体肌和肛门外括约肌在会阴体处合并。盆底结构的神经供应主要来自骶神经S3和S(作为阴部神经)。主要的血液供应来自髂内动脉的壁支。盆底肌肉有三个功能:1. 支撑盆腔器官——膀胱、尿道、前列腺(男性)、阴道和子宫(女性)、肛门和直肠,以及对腹腔内容物的总体支撑。2. 有助于尿液和粪便的控制。3. 有助于性功能中的性唤起和性高潮。由于张力亢进、张力减退、盆腔支撑结构丧失或多种因素混合,PFD可导致多种病症。泌尿系统:排尿困难:犹豫、尿流延迟。膀胱膨出:膀胱向阴道(前部)膨出或疝出。尿道膨出(尿道脱垂):尿道向阴道(前部)膨出。尿失禁:尿液不自主漏出。妇科:性交困难:性交时或性交后疼痛。子宫脱垂:子宫经阴道脱出阴道口外。阴道脱垂:阴道顶端脱出阴道口外。肠膨出:肠管向阴道(顶端/后部)膨出或疝出。直肠膨出:直肠向阴道(后部)膨出或疝出。结直肠:便秘:排便时盆底肌肉反常收缩或松弛不足(排便协同失调)。大便失禁:粪便不自主漏出(与括约肌破裂无关)。直肠脱垂:直肠套叠至肛门缘以外(完全性直肠脱垂)或肛门近端(隐匿性直肠脱垂)。一般情况:盆腔疼痛:持续超过三至六个月的慢性疼痛,与其他明确病症无关。肛提肌痉挛:与肛提肌相关的慢性盆腔疼痛的另一种说法。直肠痛性痉挛:与肛提肌相关的短暂性痉挛性疼痛。会阴下降——会阴在骨盆出口下方膨出。