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Author Correction: Increased susceptibility of airway epithelial cells from ataxia-telangiectasia to S. pneumoniae infection due to oxidative damage and impaired innate immunity.作者更正：由于氧化损伤和先天免疫受损，共济失调毛细血管扩张症患者的气道上皮细胞对肺炎链球菌感染的易感性增加。
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2
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作者更正：由于氧化损伤和先天免疫受损，共济失调毛细血管扩张症患者的气道上皮细胞对肺炎链球菌感染的易感性增加。

Author Correction: Increased susceptibility of airway epithelial cells from ataxia-telangiectasia to S. pneumoniae infection due to oxidative damage and impaired innate immunity.

作者信息

Yeo Abrey J, Henningham Anna, Fantino Emmanuelle, Galbraith Sally, Krause Lutz, Wainwright Claire E, Sly Peter D, Lavin Martin F

机构信息

Neuroscience & Infectious Disease Group, The University of Queensland Centre for Clinical Research, Herston, Queensland, Australia.

Children's Lung, Environment and Asthma Research (CLEAR) Group, Child Health Research Centre, The University of Queensland, South Brisbane, Queensland, Australia.

出版信息

Sci Rep. 2020 Jul 24;10(1):12742. doi: 10.1038/s41598-020-69649-w.

DOI:10.1038/s41598-020-69649-w
PMID:32704151
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7378188/
Abstract

An amendment to this paper has been published and can be accessed via a link at the top of the paper.

摘要

本文的一个修订版本已发表，可通过本文顶部的链接获取。