Christensen R L, Shade D L, Graves C B, McDonald J M
Int J Biochem. 1987;19(3):259-65. doi: 10.1016/0020-711x(87)90029-2.
The role of protein kinase C in the mechanism of stimulation of glucose transport in rat adipocytes was investigated. Glucose transport was stimulated by dioleoylglycerol (DOG), tetradecanoyl phorbol acetate (TPA) and phospholipase C (PLC). Agents that inhibit protein kinase C (polymyxin B, gossypol and quercitin) also inhibited glucose transport that had been stimulated by DOG, TPA, PLC and insulin.
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