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嗜铬细胞瘤破裂诱发的基底型应激性心肌病

Basal Takotsubo syndrome induced by pheochromocytoma rupture.

作者信息

Yuan Shanshan, He Tao, Yang Lijia, Chu Qiang, Huang Weiqing, Dai Hongyan

机构信息

Department of Cardiology, Qingdao Municipal Hospital, Qingdao, Shandong, China.

Department of Pathology, Qingdao Municipal Hospital, Qingdao, Shandong, China.

出版信息

Cardiovasc J Afr. 2020 Oct 5;31:1-4. doi: 10.5830/CVJA-2020-040.

Abstract

Takotsubo syndrome (TTS), characterised by transient left ventricular systolic dysfunction, is divided into five types: (1) apical ballooning, (2) mid-ventricular, (3) basal or inverted, (4) and focal wall-motion patterns, and (5) other types, including biventricular type, isolated right ventricular and global type. The common clinical features of TTS are similar to acute coronary syndrome, which makes them indistinguishable in the early stages. TTS has a wide spectrum of emotional or physical triggers. Pheochromocytoma has been widely recognised as a distinct physical trigger of TTS. Although reports of pheochromocytoma causing TTS are not uncommon, spontaneous rupture of pheochromocytoma causing TTS is extremely rare because of the low incidence of tumour rupture. Here we report on a case of a 31-year-old man with adrenal pheochromocytoma rupture developing basal TTS.

摘要

应激性心肌病(TTS),其特征为短暂性左心室收缩功能障碍,分为五种类型:(1)心尖部气球样变型,(2)心室中部型,(3)基底部或倒置型,(4)局灶性室壁运动型,以及(5)其他类型,包括双心室型、孤立性右心室型和整体型。TTS的常见临床特征与急性冠状动脉综合征相似,这使得它们在早期难以区分。TTS有多种情绪或身体触发因素。嗜铬细胞瘤已被广泛认为是TTS一种独特的身体触发因素。虽然嗜铬细胞瘤导致TTS的报道并不少见,但由于肿瘤破裂发生率低,嗜铬细胞瘤自发破裂导致TTS极为罕见。在此,我们报告一例31岁男性,因肾上腺嗜铬细胞瘤破裂并发基底部TTS。

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