Familial Mediterranean fever--an update.
作者信息
Goldfinger S E
出版信息
Trans Am Clin Climatol Assoc. 1987;98:128-34.
Abstract
摘要
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本文引用的文献
1
Leukocyte chemotaxis in recurrent polyserositis (familial Mediterranean fever).
Am J Med Sci. 1981 Jan-Feb;281(1):15-8. doi: 10.1097/00000441-198101000-00002.
2
Synthesis and secretion of serum amyloid protein A (SAA) by hepatocytes in mice treated with casein.用酪蛋白处理的小鼠肝细胞中血清淀粉样蛋白A（SAA）的合成与分泌。
J Immunol. 1980 Feb;124(2):495-9.
3
C5a-inhibitor deficiency in peritoneal fluids from patients with familial Mediterranean fever.家族性地中海热患者腹腔液中C5a抑制剂缺乏
N Engl J Med. 1984 Aug 2;311(5):287-90. doi: 10.1056/NEJM198408023110503.
4
Gastrointestinal effects of long-term colchicine therapy in patients with recurrent polyserositis (familial mediterranean fever).长期秋水仙碱治疗复发性多浆膜炎（家族性地中海热）患者的胃肠道影响。
Dig Dis Sci. 1982 Aug;27(8):723-7. doi: 10.1007/BF01393768.
5
Correction of a suppressor cell deficiency in four patients with familial Mediterranean fever by in vitro or in vivo colchicine.通过体外或体内使用秋水仙碱纠正四名家族性地中海热患者的抑制细胞缺陷。
Clin Exp Immunol. 1982 Oct;50(1):99-106.
6
Interleukin-1 and the pathogenesis of the acute-phase response.白细胞介素-1与急性期反应的发病机制。
N Engl J Med. 1984 Nov 29;311(22):1413-8. doi: 10.1056/NEJM198411293112205.
7
Hyperimmunoglobulinaemia D and periodic fever: a new syndrome.高免疫球蛋白D血症与周期性发热：一种新综合征。
Lancet. 1984 May 19;1(8386):1087-90. doi: 10.1016/s0140-6736(84)92505-4.
8
Mechanism of reduced amyloid-A-degrading activity in serum of patients with secondary amyloidosis.
Lancet. 1982 Jul 31;2(8292):234-7. doi: 10.1016/s0140-6736(82)90322-1.
9
Serum amyloid-A protein concentration in inflammatory diseases and its relationship to the incidence of reactive systemic amyloidosis.炎症性疾病中血清淀粉样蛋白A的浓度及其与反应性系统性淀粉样变性发病率的关系。
Lancet. 1982 Jul 31;2(8292):231-4. doi: 10.1016/s0140-6736(82)90321-x.
10
The amino acid sequence of a major nonimmunoglobulin component of some amyloid fibrils.某些淀粉样纤维主要非免疫球蛋白成分的氨基酸序列。
J Clin Invest. 1972 Oct;51(10):2773-6. doi: 10.1172/JCI107098.
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