Molvaer O I, Eidsvik S
Undersea Biomed Res. 1987 May;14(3):277-95.
If impaired eustachian tube function causes an overpressure to remain in the middle ear after ascent in diving or aviation in a subject with a defect in the wall of the facial canal, an ischemic neurapraxia of the seventh cranial nerve may occur. This type of facial palsy is designated facial baroparesis, baroparesis facialis, or alternobaric facial palsy. If the middle ear pressure is asymmetric the subject may also have alternobaric vertigo. A causative relationship between middle ear overpressure and facial palsy is supported by the palsy's rapid onset following a reduction in ambient pressure and by its quick disappearance after either an increase in ambient pressure or release of the middle ear overpressure. Transient compression-induced ischemic neurapraxia of the facial nerve is also demonstrated in animal experiments. A similar palsy, ischemic neurapraxia of the fifth cranial nerve due to compression in the maxillary sinus, has been reported in divers. Although it is under-reported, facial baroparesis occurs infrequently, with 23 subjects mentioned in the available literature. Nevertheless, it is important to be aware of its existence, because misdiagnosis as type II DCS or air embolism results in unnecessarily long recompression treatments and pointless delay of resumption of diving. In the worst case, a misdiagnosis might cause a diving license to be revoked.
如果咽鼓管功能受损,导致在潜水或航空上升过程中,中耳内压力在面神经管管壁有缺损的个体中持续过高,可能会发生第七颅神经的缺血性神经失用症。这种类型的面瘫被称为面部气压性轻瘫、气压性面瘫或交替性气压性面瘫。如果中耳压力不对称,个体也可能出现交替性气压性眩晕。中耳压力过高与面瘫之间的因果关系得到以下事实的支持:面瘫在环境压力降低后迅速发作,在环境压力增加或中耳压力过高解除后迅速消失。动物实验也证明了短暂的压迫性缺血性面神经神经失用症。潜水员中曾报告过类似的面瘫,即由于上颌窦受压导致的第五颅神经缺血性神经失用症。尽管报道较少,但面部气压性轻瘫很少发生,现有文献中仅提及23例。然而,了解其存在很重要,因为误诊为II型减压病或空气栓塞会导致不必要的长时间再加压治疗以及潜水恢复的无意义延迟。在最坏的情况下,误诊可能导致潜水执照被吊销。