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The interplay between apoptosis and ferroptosis mediated by ER stress.

作者信息

Lee Yong J

机构信息

Department of Surgery, School of Medicine, University of Pittsburgh, 5117 Centre Ave, Pittsburgh, PA, 15213, USA.

出版信息

Apoptosis. 2020 Dec;25(11-12):783. doi: 10.1007/s10495-020-01642-0. Epub 2020 Nov 2.

DOI:10.1007/s10495-020-01642-0
PMID:33140179
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7680372/
Abstract
摘要

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本文引用的文献

1
BAX-dependent mitochondrial pathway mediates the crosstalk between ferroptosis and apoptosis.BAX 依赖性线粒体途径介导铁死亡与细胞凋亡之间的串扰。
Apoptosis. 2020 Oct;25(9-10):625-631. doi: 10.1007/s10495-020-01627-z.
2
Analysis of the Mitochondrial Membrane Potential Using the Cationic JC-1 Dye as a Sensitive Fluorescent Probe.使用阳离子型JC-1染料作为灵敏荧光探针分析线粒体膜电位
Bio Protoc. 2019 Jan 5;9(1). doi: 10.21769/BioProtoc.3128.
3
SCF(FBW7) regulates cellular apoptosis by targeting MCL1 for ubiquitylation and destruction.SCF（FBW7）通过靶向 MCL1 进行泛素化和降解来调节细胞凋亡。
Nature. 2011 Mar 3;471(7336):104-9. doi: 10.1038/nature09732.
4
Caspase-mediated cleavage of Beclin-1 inactivates Beclin-1-induced autophagy and enhances apoptosis by promoting the release of proapoptotic factors from mitochondria.Caspase 介导的 Beclin-1 切割使 Beclin-1 诱导的自噬失活，并通过促进促凋亡因子从线粒体释放来增强细胞凋亡。
Cell Death Dis. 2010;1(1):e18. doi: 10.1038/cddis.2009.16.
5
Phosphorylation and inactivation of myeloid cell leukemia 1 by JNK in response to oxidative stress.在氧化应激反应中，JNK导致髓样细胞白血病1磷酸化并使其失活。
J Biol Chem. 2002 Nov 15;277(46):43730-4. doi: 10.1074/jbc.M207951200. Epub 2002 Sep 9.