Reply to Picod : Alteration of the Renin-Angiotensin-Aldosterone System in Shock: Role of the Dipeptidyl Peptidase 3.
作者信息
Chawla Lakhmir S, Bellomo Rinaldo
机构信息
Veterans Affairs Medical Center San Diego, California and.
The University of Melbourne Melbourne, Victoria, Australia.
出版信息
Am J Respir Crit Care Med. 2021 Feb 15;203(4):527-528. doi: 10.1164/rccm.202010-3968LE.
Abstract
摘要
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Reply to Picod : Alteration of the Renin-Angiotensin-Aldosterone System in Shock: Role of the Dipeptidyl Peptidase 3.对皮科德的回复：休克中肾素-血管紧张素-醛固酮系统的改变：二肽基肽酶3的作用
Am J Respir Crit Care Med. 2021 Feb 15;203(4):527-528. doi: 10.1164/rccm.202010-3968LE.
2
Alteration of the Renin-Angiotensin-Aldosterone System in Shock: Role of the Dipeptidyl Peptidase 3.休克时肾素-血管紧张素-醛固酮系统的改变：二肽基肽酶3的作用
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本文引用的文献
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Impaired angiotensin II type 1 receptor signaling contributes to sepsis-induced acute kidney injury.血管紧张素 II 型 1 受体信号转导受损导致脓毒症引起的急性肾损伤。
Kidney Int. 2021 Jan;99(1):148-160. doi: 10.1016/j.kint.2020.07.047. Epub 2020 Aug 31.
2
Renin and Survival in Patients Given Angiotensin II for Catecholamine-Resistant Vasodilatory Shock. A Clinical Trial.血管扩张性休克儿茶酚胺抵抗患者应用血管紧张素 II 治疗与肾素及生存关系的临床研究
Am J Respir Crit Care Med. 2020 Nov 1;202(9):1253-1261. doi: 10.1164/rccm.201911-2172OC.
3
Angiotensin II in experimental hyperdynamic sepsis.实验性高动力败血症中的血管紧张素 II。
Crit Care. 2009;13(6):R190. doi: 10.1186/cc8185. Epub 2009 Nov 30.
4
Beta-arrestin2-mediated inotropic effects of the angiotensin II type 1A receptor in isolated cardiac myocytes.β-抑制蛋白2介导的1A型血管紧张素受体在离体心肌细胞中的变力作用。
Proc Natl Acad Sci U S A. 2006 Oct 31;103(44):16284-9. doi: 10.1073/pnas.0607583103. Epub 2006 Oct 23.
5
Molecular mechanisms of angiotensin II receptor internalization.血管紧张素II受体内化的分子机制。
J Am Soc Nephrol. 1999 Jan;10 Suppl 11:S47-56.
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