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本文引用的文献

1
Targeting DNA Methylation Depletes Uterine Leiomyoma Stem Cell-enriched Population by Stimulating Their Differentiation.靶向 DNA 甲基化通过刺激其分化来耗竭富含子宫肌瘤干细胞的群体。
Endocrinology. 2020 Oct 1;161(10). doi: 10.1210/endocr/bqaa143.
2
RANKL/RANK Pathway and Its Inhibitor RANK-Fc in Uterine Leiomyoma Growth.RANKL/RANK 通路及其抑制剂 RANK-Fc 在子宫肌瘤生长中的作用。
J Clin Endocrinol Metab. 2018 May 1;103(5):1842-1849. doi: 10.1210/jc.2017-01585.
3
Human uterine leiomyoma stem/progenitor cells expressing CD34 and CD49b initiate tumors in vivo.表达CD34和CD49b的人子宫平滑肌瘤干/祖细胞在体内引发肿瘤。
J Clin Endocrinol Metab. 2015 Apr;100(4):E601-6. doi: 10.1210/jc.2014-2134. Epub 2015 Feb 6.
4
Progesterone is essential for maintenance and growth of uterine leiomyoma.孕激素对于维持和生长子宫肌瘤是必需的。
Endocrinology. 2010 Jun;151(6):2433-42. doi: 10.1210/en.2009-1225. Epub 2010 Apr 7.
5
Uterine fibroid tumors: diagnosis and treatment.子宫肌瘤：诊断与治疗
Am Fam Physician. 2007 May 15;75(10):1503-8.
6
The frequency of uterine leiomyomas.子宫平滑肌瘤的发病率。
Am J Clin Pathol. 1990 Oct;94(4):435-8. doi: 10.1093/ajcp/94.4.435.

Towards an Epigenetic Treatment of Leiomyomas?

作者信息

Vaiman Daniel

机构信息

Department of Development, Reproduction and Cancer, Cochin Institute, INSERM, CNRS, Université de Paris, Paris, France.

出版信息

Endocrinology. 2020 Dec 1;161(12). doi: 10.1210/endocr/bqaa172.

DOI:10.1210/endocr/bqaa172
PMID:33156897
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7646627/
Abstract
摘要