体外诱导人和啮齿动物胰岛实质细胞上的II类主要组织相容性复合体抗原 - Suppr

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Induction of class II MHC antigens on human and rodent islet parenchymal cells in vitro.

作者信息

Wright J R, Lacy P E, Unanue E R, Hauptfeld V

出版信息

Diabetologia. 1987 Jun;30(6):441. doi: 10.1007/BF00292550.

DOI:10.1007/BF00292550

PMID:3315799

Abstract

摘要

相似文献

Induction of class II MHC antigens on human and rodent islet parenchymal cells in vitro.体外诱导人和啮齿动物胰岛实质细胞上的II类主要组织相容性复合体抗原

Diabetologia. 1987 Jun;30(6):441. doi: 10.1007/BF00292550.

Human islet preparations and the expression of MHC antigens.人胰岛制剂与主要组织相容性复合体（MHC）抗原的表达

Transplant Proc. 1992 Jun;24(3):1029.

Induction of class II MHC antigens in vitro on pancreatic B cells isolated from BB/E rats.从BB/E大鼠分离的胰腺β细胞体外II类主要组织相容性复合体抗原的诱导

Diabetologia. 1986 Oct;29(10):749-51. doi: 10.1007/BF00870286.

Control of islet intercellular adhesion molecule-1 expression by interferon-alpha and hypoxia.

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Expression of HLA in isolated human pancreatic islets and cryopreservation.

Transplant Proc. 1987 Feb;19(1 Pt 1):220-1.

Synergy of class I and class II MHC deficiency (C1D and C2D) islets and early short-term suppression in achieving long-term islet graft survival and tolerance induction.I类和II类主要组织相容性复合体缺陷（C1D和C2D）胰岛的协同作用以及早期短期抑制在实现长期胰岛移植存活和诱导耐受性方面的作用。

Transplant Proc. 1999 Feb-Mar;31(1-2):635-6. doi: 10.1016/s0041-1345(98)01591-7.

MHC antigen induction by interferon gamma on cultured mouse pancreatic beta cells and macrophages. Genetic analysis of strain differences and discovery of an "occult" class I-like antigen in NOD/Lt mice.干扰素γ对培养的小鼠胰腺β细胞和巨噬细胞的MHC抗原诱导作用。品系差异的遗传分析以及在NOD/Lt小鼠中发现一种“隐匿”的I类样抗原。

J Exp Med. 1989 Oct 1;170(4):1243-62. doi: 10.1084/jem.170.4.1243.

[Presence and manipulation of MHC class II antigens on isolated island of Langerhans cells].

Z Exp Chir Transplant Kunstliche Organe. 1986;19(4):247-9.

Differential expression and regulation of MHC products in the endocrine and exocrine cells of the human pancreas.人类胰腺内分泌和外分泌细胞中主要组织相容性复合体（MHC）产物的差异表达与调控

Clin Exp Immunol. 1986 Jul;65(1):128-39.

HLA class II induction in human islet cells by interferon-gamma plus tumour necrosis factor or lymphotoxin.干扰素-γ加肿瘤坏死因子或淋巴毒素诱导人胰岛细胞表达HLA-II类分子。

Nature. 1987;326(6110):304-6. doi: 10.1038/326304a0.

引用本文的文献

Tumor necrosis factor enhances interferon-induced Ia antigen expression on murine islet parenchymal cells.肿瘤坏死因子增强干扰素诱导的小鼠胰岛实质细胞Ia抗原表达。

Am J Pathol. 1988 Mar;130(3):427-30.

Induction of Ia antigen expression on murine islet parenchymal cells does not diminish islet allograft survival.诱导小鼠胰岛实质细胞表达Ia抗原不会降低胰岛同种异体移植的存活率。

Am J Pathol. 1989 Feb;134(2):237-42.

Identification of cells responsible for the suppression of anti-beta-cell cytotoxicity in type I (insulin-dependent) diabetes mellitus.鉴定在I型（胰岛素依赖型）糖尿病中负责抑制抗β细胞细胞毒性的细胞。

Clin Exp Immunol. 1991 Apr;84(1):43-7. doi: 10.1111/j.1365-2249.1991.tb08121.x.

本文引用的文献

Beta-cell damage in diabetic insulitis: are we approaching a solution?糖尿病性胰岛炎中的β细胞损伤：我们正在接近解决方案吗？

Diabetologia. 1984 Apr;26(4):241-9. doi: 10.1007/BF00283644.

Interferon-gamma enhances the expression of the major histocompatibility class I antigens on mouse pancreatic beta cells.γ干扰素增强小鼠胰腺β细胞上主要组织相容性复合体I类抗原的表达。

Diabetes. 1985 Nov;34(11):1205-9. doi: 10.2337/diab.34.11.1205.

Pre-diabetes in the spontaneously diabetic BB/E rat: lymphocyte subpopulations in the pancreatic infiltrate and expression of rat MHC class II molecules in endocrine cells.

Diabetologia. 1985 Jul;28(7):464-6. doi: 10.1007/BF00280892.

Induction of class II MHC antigens in vitro on pancreatic B cells isolated from BB/E rats.从BB/E大鼠分离的胰腺β细胞体外II类主要组织相容性复合体抗原的诱导

Diabetologia. 1986 Oct;29(10):749-51. doi: 10.1007/BF00870286.

HLA class II induction in human islet cells by interferon-gamma plus tumour necrosis factor or lymphotoxin.干扰素-γ加肿瘤坏死因子或淋巴毒素诱导人胰岛细胞表达HLA-II类分子。

Nature. 1987;326(6110):304-6. doi: 10.1038/326304a0.

Clin Exp Immunol. 1986 Jul;65(1):128-39.

Interferon-mediated induction of Ia antigen expression on isolated murine whole islets and dispersed islet cells.干扰素介导的分离小鼠全胰岛和分散胰岛细胞上Ia抗原表达的诱导。

Diabetes. 1986 Oct;35(10):1174-7. doi: 10.2337/diab.35.10.1174.

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