肾移植后的骨病、低磷血症和甲状旁腺功能亢进
Bone disease, hypophosphatemia and hyperparathyroidism after renal transplantation.
作者信息
Nielsen H E, Christensen M S, Melsen F, Torring S
出版信息
Adv Exp Med Biol. 1977;81:603-10. doi: 10.1007/978-1-4613-4217-5_58.
UNLABELLED
Long-term survivors after renal transplantation (RT) show: 1) decreased s-phosphorus 2) increased s-PTH 3) no correlation between s-PTH and s-phosphorus 4) increased amount of non-mineralized bone (osteomalacia), inversely correlated to s-phosphorus 5) decreased amount of trabecular bone (osteopenia) in RT patients with aseptic necorsis of bone.
CONCLUSION
aseptic necrosis of bone after RT is mainly due to osteoid-induced osteopenia, perhaps aggravated by hypophosphatemic osteomalacia.
未标记
肾移植（RT）后的长期存活者表现为：1）血清磷降低；2）血清甲状旁腺激素升高；3）血清甲状旁腺激素与血清磷之间无相关性；4）非矿化骨量增加（骨软化症），与血清磷呈负相关；5）在发生无菌性骨坏死的肾移植患者中，小梁骨量减少（骨质减少）。
结论
肾移植后无菌性骨坏死主要归因于类骨质引起的骨质减少，低磷性骨软化症可能会使其加重。
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