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PPARs and Myocardial Infarction.过氧化物酶体增殖物激活受体与心肌梗死。
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PPARs and Angiogenesis-Implications in Pathology.过氧化物酶体增殖物激活受体与血管生成——病理学意义。
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Peroxisome proliferator-activated receptors and inflammation.过氧化物酶体增殖物激活受体与炎症
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Pharmacological Utility of PPAR Modulation for Angiogenesis in Cardiovascular Disease.PPAR 调节在心血管疾病血管生成中的药理学作用。
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Peroxisome proliferator-activated receptor (PPAR)β/δ, a possible nexus of PPARα- and PPARγ-dependent molecular pathways in neurodegenerative diseases: Review and novel hypotheses.过氧化物酶体增殖物激活受体 (PPAR)β/δ，神经退行性疾病中 PPARα 和 PPARγ 依赖的分子途径的可能连接点：综述和新假说。
Neurochem Int. 2013 Oct;63(4):322-30. doi: 10.1016/j.neuint.2013.06.012. Epub 2013 Jun 25.
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The peroxisome proliferator-activated receptors in cardiovascular diseases: experimental benefits and clinical challenges.心血管疾病中的过氧化物酶体增殖物激活受体：实验益处与临床挑战
Br J Pharmacol. 2015 Dec;172(23):5512-22. doi: 10.1111/bph.13029. Epub 2015 Jan 23.
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Multifaceted roles of peroxisome proliferator-activated receptors (PPARs) at the cellular and whole organism levels.过氧化物酶体增殖物激活受体 (PPARs) 在细胞和整体水平上的多方面作用。
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Emerging roles of peroxisome proliferator-activated receptor-beta/delta in inflammation.过氧化物酶体增殖物激活受体-β/δ 在炎症中的新兴作用。
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The Role of PPARs in Disease.过氧化物酶体增殖物激活受体（PPARs）在疾病中的作用。
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International Union of Pharmacology. LXI. Peroxisome proliferator-activated receptors.国际药理学联合会。LX I. 过氧化物酶体增殖物激活受体。
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Nuclear Receptor ERRγ Protects Against Cardiac Ischemic Injury by Suppressing GBP5-Mediated Myocardial Inflammation.核受体ERRγ通过抑制GBP5介导的心肌炎症来预防心脏缺血性损伤。
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Identification of lipid metabolism-related genes in myocardial infarction: implications for diagnosis and therapy.心肌梗死中脂质代谢相关基因的鉴定：对诊断和治疗的意义。
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Interplay between energy metabolism and NADPH oxidase-mediated pathophysiology in cardiovascular diseases.心血管疾病中能量代谢与NADPH氧化酶介导的病理生理学之间的相互作用。
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Pleiotropic Effects of Peroxisome Proliferator-Activated Receptor Alpha and Gamma Agonists on Myocardial Damage: Molecular Mechanisms and Clinical Evidence-A Narrative Review.过氧化物酶体增殖物激活受体α和γ激动剂对心肌损伤的多效作用：分子机制和临床证据——叙述性综述。
Cells. 2024 Sep 5;13(17):1488. doi: 10.3390/cells13171488.
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PPARs in Clinical Experimental Medicine after 35 Years of Worldwide Scientific Investigations and Medical Experiments.经过 35 年的全球科学研究和医学实验，PPARs 在临床实验医学中的应用。
Biomolecules. 2024 Jul 1;14(7):786. doi: 10.3390/biom14070786.
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VSP-2 attenuates secretion of inflammatory cytokines induced by LPS in BV2 cells by mediating the PPARγNF-κB signaling pathway.VSP-2通过介导PPARγ-NF-κB信号通路减弱脂多糖诱导的BV2细胞中炎性细胞因子的分泌。
Open Life Sci. 2024 Apr 20;19(1):20220861. doi: 10.1515/biol-2022-0861. eCollection 2024.
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Valerenic acid attenuates pathological myocardial hypertrophy by promoting the utilization of multiple substrates in the mitochondrial energy metabolism.缬草烯酸通过促进线粒体能量代谢中多种底物的利用来减轻病理性心肌肥大。
J Adv Res. 2025 Feb;68:241-256. doi: 10.1016/j.jare.2024.02.008. Epub 2024 Feb 18.
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Network Pharmacology and Experimental Validation to Explore Mechanism of Tetrahydropalmatine on Acute Myocardial Ischemia.网络药理学与实验验证探索延胡索乙素治疗急性心肌缺血的作用机制。
Chin J Integr Med. 2023 Dec;29(12):1087-1098. doi: 10.1007/s11655-023-3644-x. Epub 2023 Aug 22.
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Pharmacological Utility of PPAR Modulation for Angiogenesis in Cardiovascular Disease.PPAR 调节在心血管疾病血管生成中的药理学作用。
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Peroxisome Proliferator-Activated Receptors and the Hallmarks of Cancer.过氧化物酶体增殖物激活受体与癌症的特征。
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本文引用的文献
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The molecular mechanism involved in cardioprotection by the dietary flavonoid fisetin as an agonist of PPAR-γ in a murine model of myocardial infarction.膳食类黄酮漆黄素通过激活 PPAR-γ 对心肌梗死小鼠发挥心脏保护作用的分子机制研究。
Arch Biochem Biophys. 2020 Nov 15;694:108572. doi: 10.1016/j.abb.2020.108572. Epub 2020 Sep 12.
2
PPARs and Angiogenesis-Implications in Pathology.过氧化物酶体增殖物激活受体与血管生成——病理学意义。
Int J Mol Sci. 2020 Aug 10;21(16):5723. doi: 10.3390/ijms21165723.
3
Chrysin rescues rat myocardium from ischemia-reperfusion injury via PPAR-γ/Nrf2 activation.白杨素通过激活 PPAR-γ/Nrf2 拯救大鼠心肌缺血再灌注损伤。
Eur J Pharmacol. 2020 Sep 15;883:173389. doi: 10.1016/j.ejphar.2020.173389. Epub 2020 Jul 22.
4
PPAR Beta/Delta and the Hallmarks of Cancer.过氧化物酶体增殖物激活受体-β/δ 与癌症的特征。
Cells. 2020 May 4;9(5):1133. doi: 10.3390/cells9051133.
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Vascular PPARβ/δ Promotes Tumor Angiogenesis and Progression.血管 PPARβ/δ 促进肿瘤血管生成和进展。
Cells. 2019 Dec 12;8(12):1623. doi: 10.3390/cells8121623.
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Ginsenoside Rb3 regulates energy metabolism and apoptosis in cardiomyocytes via activating PPARα pathway.人参皂苷 Rb3 通过激活 PPARα 通路调节心肌细胞能量代谢和细胞凋亡。
Biomed Pharmacother. 2019 Dec;120:109487. doi: 10.1016/j.biopha.2019.109487. Epub 2019 Sep 29.
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Theacrine attenuates myocardial fibrosis after myocardial infarction via the SIRT3/β-catenin/PPARγ pathway in estrogen-deficient mice.咖啡酰奎尼酸通过 SIRT3/β-catenin/PPARγ 通路减轻去势雌鼠心肌梗死后的心肌纤维化。
Eur Rev Med Pharmacol Sci. 2019 Jun;23(12):5477-5486. doi: 10.26355/eurrev_201906_18217.
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Pioglitazone strengthen therapeutic effect of adipose-derived regenerative cells against ischemic cardiomyopathy through enhanced expression of adiponectin and modulation of macrophage phenotype.吡格列酮通过增强脂联素表达和调节巨噬细胞表型增强脂肪来源的再生细胞对缺血性心肌病的治疗效果。
Cardiovasc Diabetol. 2019 Mar 22;18(1):39. doi: 10.1186/s12933-019-0829-x.
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Clofibrate Treatment Decreases Inflammation and Reverses Myocardial Infarction-Induced Remodelation in a Rodent Experimental Model.氯贝丁酯治疗可减少炎症反应并逆转啮齿动物实验模型中心肌梗死引起的重构。
Molecules. 2019 Jan 12;24(2):270. doi: 10.3390/molecules24020270.
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PPARβ/δ a potential target in pulmonary hypertension blighted by cancer risk.PPARβ/δ：癌症风险困扰下肺动脉高压的一个潜在靶点。
Pulm Circ. 2019 Jan-Mar;9(1):2045894018812053. doi: 10.1177/2045894018812053. Epub 2018 Oct 23.
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