Shore D, Miles C A
AFRC Institute of Food Research, Bristol Laboratory, Langford, Avon, UK.
Ultrasonics. 1988 Jan;26(1):31-6. doi: 10.1016/0041-624x(88)90046-7.
It has been suggested that viscous losses, caused by the motion of myofibrils relative to their environmental fluid, could be the major cause of ultrasound attenuation in muscle. This Paper presents theoretical and experimental estimates of the viscous component of attenuation in suspensions of myofibrils. Experimental estimates were made by monitoring the effects of varying the viscosity and density of the suspending fluid and ranged from 0.55 to 0.72 cm2 g-1 protein at 7 MHz, when expressed as mass attenuation coefficients. This represented 16-22% of the total attenuation measured in suspensions. Corresponding theoretical calculations were lower: 0.44 and 0.21 cm2 g-1 protein, respectively, for suspensions with the myofibrils aligned along and across the direction of sound propagation. It was concluded that most of the attenuation was caused by other absorption processes.
有人提出，肌原纤维相对于其周围流体的运动所引起的粘性损失，可能是肌肉中超声衰减的主要原因。本文给出了肌原纤维悬浮液中衰减粘性成分的理论和实验估计值。实验估计是通过监测改变悬浮液的粘度和密度的影响来进行的，当以质量衰减系数表示时，在7兆赫下范围为0.55至0.72平方厘米每克蛋白质。这占悬浮液中测得的总衰减的16 - 22%。相应的理论计算值较低：对于肌原纤维沿声传播方向排列和垂直于声传播方向排列的悬浮液，分别为0.44和0.21平方厘米每克蛋白质。得出的结论是，大部分衰减是由其他吸收过程引起的。
