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A disease mutation reveals a role for NaV1.9 in acute itch.
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Voltage-Gated Sodium Channels Regulating Action Potential Generation in Itch-, Nociceptive-, and Low-Threshold Mechanosensitive Cutaneous C-Fibers.
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Mas-Related G Protein-Coupled Receptors and the Biology of Itch Sensation.
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Targeting human Mas-related G protein-coupled receptor X1 to inhibit persistent pain.
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Mechanisms of pruritogen-induced activation of itch nerves in isolated mouse skin.
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The phenotype of congenital insensitivity to pain due to the NaV1.9 variant p.L811P.
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Unbiased classification of sensory neuron types by large-scale single-cell RNA sequencing.
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The role of slow and persistent TTX-resistant sodium currents in acute tumor necrosis factor-α-mediated increase in nociceptors excitability.
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Discovery and characterization of 2-(cyclopropanesulfonamido)-N-(2-ethoxyphenyl)benzamide, ML382: a potent and selective positive allosteric modulator of MrgX1.
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