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巨大心脏黏液瘤致劳力性心绞痛:新生血管导致的冠状动脉窃血?

Giant cardiac myxoma presenting with exertional angina: Coronary steal due to neovascularization?

机构信息

Department of Cardiac Surgery, Zhongshan Hospital Fudan University, Shanghai, China.

出版信息

J Card Surg. 2021 Mar;36(3):1134-1135. doi: 10.1111/jocs.15366. Epub 2021 Jan 21.

DOI:10.1111/jocs.15366
PMID:33476471
Abstract

Myxoma is the most common benign tumor of the heart. Most patients present with no symptoms, only a few patients present with exertional dyspnea and stroke. We introduce this rare case presenting with exertional angina, which was caused by coronary steal due to neovascularization, proved by coronary angiography and cardiac stress testing.

摘要

黏液瘤是心脏最常见的良性肿瘤。大多数患者无症状,少数患者出现劳力性呼吸困难和卒中。我们介绍这例罕见的劳力性心绞痛病例,其由冠状动脉盗血引起,原因是新生血管化导致的冠状动脉造影和心脏负荷试验证实。

相似文献

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Giant cardiac myxoma presenting with exertional angina: Coronary steal due to neovascularization?巨大心脏黏液瘤致劳力性心绞痛:新生血管导致的冠状动脉窃血?
J Card Surg. 2021 Mar;36(3):1134-1135. doi: 10.1111/jocs.15366. Epub 2021 Jan 21.
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Cystic and hemorrhagic giant left atrial myxoma in a patient presenting with exertional angina and dyspnea.患者因劳力性心绞痛和呼吸困难就诊,发现左侧心房粘液瘤囊性和出血性巨大。
Cardiovasc Pathol. 2012 Mar-Apr;21(2):e15-8. doi: 10.1016/j.carpath.2011.01.006. Epub 2011 Mar 11.
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Neovascularization of floating myxoma clearly depicted on angiograms.血管造影清晰显示漂浮性黏液瘤的新生血管形成。
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The diagnosis and clinical importance of tumour neovascularisation from coronary artery to right atrial myxoma.从冠状动脉到右心房黏液瘤的肿瘤新生血管的诊断和临床意义。
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Left atrial myxoma causing coronary steal: an atypical cause of angina.左心房黏液瘤导致冠状动脉窃血:心绞痛的非典型病因。
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Neovascularized myxoma-causing abnormal blood flow in the left atrium diagnosed by transesophageal echocardiography.经食管超声心动图诊断出的导致左心房异常血流的血管化黏液瘤。
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Hemorrhage and associated fistula formation in a left atrial myxoma causing coronary artery steal syndrome.左心房黏液瘤伴出血及相关瘘管形成导致冠状动脉窃血综合征。
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引用本文的文献

1
Case report: Left atrial myxoma with morphology of cavernous hemangioma supplied by the right coronary artery.病例报告:具有海绵状血管瘤形态且由右冠状动脉供血的左心房黏液瘤。
Front Cardiovasc Med. 2023 Aug 1;10:1207339. doi: 10.3389/fcvm.2023.1207339. eCollection 2023.