培养的皮肤成纤维细胞中yRNA3上调以及硬皮病患者血清中yRNA4下调。 - Suppr

yRNA3 up-regulation in cultured dermal fibroblasts and yRNA4 down-regulation in the sera of scleroderma patients.

作者信息

Tani Sayaka, Kaminaka Chikako, Nishiyama Kosuke, Yariyama Azusa, Nakatani Yumi, Inaba Yutaka, Kunimoto Kayo, Yamamoto Yuki, Makino Katsunari, Makino Takamitsu, Jinnin Masatoshi

机构信息

Department of Dermatology, Wakayama Medical University, Wakayama, Japan.

出版信息

J Dermatol Sci. 2021 Apr;102(1):68-71. doi: 10.1016/j.jdermsci.2021.01.006. Epub 2021 Jan 21.

DOI:10.1016/j.jdermsci.2021.01.006

PMID:33516635

Abstract

摘要

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yRNA3 up-regulation in cultured dermal fibroblasts and yRNA4 down-regulation in the sera of scleroderma patients.培养的皮肤成纤维细胞中yRNA3上调以及硬皮病患者血清中yRNA4下调。

J Dermatol Sci. 2021 Apr;102(1):68-71. doi: 10.1016/j.jdermsci.2021.01.006. Epub 2021 Jan 21.

Mitogenic activities for skin fibroblasts in the sera from untreated scleroderma patients at the early stage.早期未经治疗的硬皮病患者血清中对皮肤成纤维细胞的促有丝分裂活性。

Arch Dermatol Res. 1989;281(6):383-6. doi: 10.1007/BF00455321.

CXCL17-mediated downregulation of type I collagen via MMP1 and miR-29 in skin fibroblasts possibly contributes to the fibrosis in systemic sclerosis.在皮肤成纤维细胞中，CXCL17通过基质金属蛋白酶1（MMP1）和微小RNA-29（miR-29）介导的I型胶原蛋白下调可能导致系统性硬化症中的纤维化。

J Dermatol Sci. 2020 Dec;100(3):183-191. doi: 10.1016/j.jdermsci.2020.09.010. Epub 2020 Sep 29.

Reduced type I collagen gene expression by skin fibroblasts of patients with systemic sclerosis after one treatment course with rituximab.利妥昔单抗一个疗程治疗后，系统性硬化症患者皮肤成纤维细胞中I型胶原蛋白基因表达降低。

Clin Exp Rheumatol. 2015 Jul-Aug;33(4 Suppl 91):S160-7. Epub 2015 Sep 1.

Increased expression of aquaporin-1 in dermal fibroblasts and dermal microvascular endothelial cells possibly contributes to skin fibrosis and edema in patients with systemic sclerosis.水通道蛋白-1 在皮肤成纤维细胞和真皮微血管内皮细胞中的表达增加可能导致系统性硬皮病患者的皮肤纤维化和水肿。

J Dermatol Sci. 2019 Jan;93(1):24-32. doi: 10.1016/j.jdermsci.2018.09.007. Epub 2018 Sep 21.

Long non-coding RNA HOTAIR drives EZH2-dependent myofibroblast activation in systemic sclerosis through miRNA 34a-dependent activation of NOTCH.长链非编码 RNA HOTAIR 通过 miRNA 34a 依赖性激活 NOTCH 驱动系统性硬化症中 EZH2 依赖性肌成纤维细胞激活。

Ann Rheum Dis. 2020 Apr;79(4):507-517. doi: 10.1136/annrheumdis-2019-216542. Epub 2020 Feb 10.

Scleroderma keratinocytes promote fibroblast activation independent of transforming growth factor beta.硬皮病角质形成细胞可促进成纤维细胞活化，且不依赖于转化生长因子β 。

Rheumatology (Oxford). 2017 Nov 1;56(11):1970-1981. doi: 10.1093/rheumatology/kex280.

miR-150 down-regulation contributes to the constitutive type I collagen overexpression in scleroderma dermal fibroblasts via the induction of integrin β3.miR-150 的下调通过诱导整合素 β3 导致硬皮病皮肤成纤维细胞中 I 型胶原的组成性过表达。

Am J Pathol. 2013 Jan;182(1):206-16. doi: 10.1016/j.ajpath.2012.09.023. Epub 2012 Nov 14.

Regulatory mechanisms of collagen expression by interleukin-22 signaling in scleroderma fibroblasts.白细胞介素-22 信号在硬皮病成纤维细胞中胶原蛋白表达的调控机制。

J Dermatol Sci. 2018 Apr;90(1):52-59. doi: 10.1016/j.jdermsci.2017.12.017. Epub 2017 Dec 29.

Persistence of scleroderma-like phenotype in normal fibroblasts after prolonged exposure to soluble mediators from mononuclear cells.

Arthritis Rheum. 1986 Jan;29(1):54-64. doi: 10.1002/art.1780290108.

引用本文的文献

Expression of collagen-related piRNA is dysregulated in cultured dermal fibroblasts derived from patients with scleroderma.硬皮病患者来源的培养真皮成纤维细胞中，胶原蛋白相关的piRNA表达失调。

Intractable Rare Dis Res. 2023 Nov;12(4):241-245. doi: 10.5582/irdr.2023.01056.

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yRNA3 up-regulation in cultured dermal fibroblasts and yRNA4 down-regulation in the sera of scleroderma patients.

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