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1
Loss of CASK Accelerates Heart Failure Development.
Circ Res. 2021 Apr 16;128(8):1139-1155. doi: 10.1161/CIRCRESAHA.120.318170. Epub 2021 Feb 17.
2
Activation of Na+/H+ exchanger 1 is sufficient to generate Ca2+ signals that induce cardiac hypertrophy and heart failure.
Circ Res. 2008 Oct 10;103(8):891-9. doi: 10.1161/CIRCRESAHA.108.175141. Epub 2008 Sep 5.
3
Targeting sarcoplasmic reticulum Ca2+ uptake to improve heart failure: hit or miss.
Circ Res. 2010 Feb 5;106(2):230-3. doi: 10.1161/CIRCRESAHA.109.210740.
6
GRK5 Controls SAP97-Dependent Cardiotoxic β Adrenergic Receptor-CaMKII Signaling in Heart Failure.
Circ Res. 2020 Aug 28;127(6):796-810. doi: 10.1161/CIRCRESAHA.119.316319. Epub 2020 Jun 8.
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Role of CaMKIIdelta phosphorylation of the cardiac ryanodine receptor in the force frequency relationship and heart failure.
Proc Natl Acad Sci U S A. 2010 Jun 1;107(22):10274-9. doi: 10.1073/pnas.1005843107. Epub 2010 May 17.

引用本文的文献

1
The Physiological and Pathological Mechanisms of LIN2, LIN7, LIN10 and Their Tripartite Complex.
J Cell Mol Med. 2025 Aug;29(15):e70794. doi: 10.1111/jcmm.70794.
2
Cardioprotective effects of semaglutide on isolated human ventricular myocardium.
Eur J Heart Fail. 2025 Mar 19. doi: 10.1002/ejhf.3644.
4
The biological functions and pathological mechanisms of CASK in various diseases.
Heliyon. 2024 Mar 29;10(8):e28863. doi: 10.1016/j.heliyon.2024.e28863. eCollection 2024 Apr 30.
5
Ion channel trafficking implications in heart failure.
Front Cardiovasc Med. 2024 Feb 14;11:1351496. doi: 10.3389/fcvm.2024.1351496. eCollection 2024.
6
Diverse Clinical Phenotypes of -Related Disorders and Multiple Functional Domains of CASK Protein.
Genes (Basel). 2023 Aug 20;14(8):1656. doi: 10.3390/genes14081656.
7
Mosaicism of a Truncating Variant of Causes Congenital Heart Disease and Neurodevelopmental Disorder.
Mol Syndromol. 2023 Jan;13(6):517-521. doi: 10.1159/000524375. Epub 2022 May 5.
8
An Overview of the Cardiorenal Protective Mechanisms of SGLT2 Inhibitors.
Int J Mol Sci. 2022 Mar 26;23(7):3651. doi: 10.3390/ijms23073651.
10
Cardiac Fibrosis Is a Risk Factor for Severe COVID-19.
Front Immunol. 2021 Oct 22;12:740260. doi: 10.3389/fimmu.2021.740260. eCollection 2021.

本文引用的文献

2
Dulaglutide and cardiovascular outcomes in type 2 diabetes (REWIND): a double-blind, randomised placebo-controlled trial.
Lancet. 2019 Jul 13;394(10193):121-130. doi: 10.1016/S0140-6736(19)31149-3. Epub 2019 Jun 9.
4
Dysferlin mediates membrane tubulation and links T-tubule biogenesis to muscular dystrophy.
J Cell Sci. 2017 Mar 1;130(5):841-852. doi: 10.1242/jcs.198861. Epub 2017 Jan 19.
5
Lateral Membrane-Specific MAGUK CASK Down-Regulates NaV1.5 Channel in Cardiac Myocytes.
Circ Res. 2016 Aug 5;119(4):544-56. doi: 10.1161/CIRCRESAHA.116.309254. Epub 2016 Jun 30.
6
Liraglutide and Cardiovascular Outcomes in Type 2 Diabetes.
N Engl J Med. 2016 Jul 28;375(4):311-22. doi: 10.1056/NEJMoa1603827. Epub 2016 Jun 13.
7
CaMKII is a RIP3 substrate mediating ischemia- and oxidative stress-induced myocardial necroptosis.
Nat Med. 2016 Feb;22(2):175-82. doi: 10.1038/nm.4017. Epub 2016 Jan 4.
8
Oxidant stress promotes disease by activating CaMKII.
J Mol Cell Cardiol. 2015 Dec;89(Pt B):160-7. doi: 10.1016/j.yjmcc.2015.10.014. Epub 2015 Oct 22.
9
S-Nitrosylation Induces Both Autonomous Activation and Inhibition of Calcium/Calmodulin-dependent Protein Kinase II δ.
J Biol Chem. 2015 Oct 16;290(42):25646-56. doi: 10.1074/jbc.M115.650234. Epub 2015 Aug 27.
10
Enhanced late INa induces proarrhythmogenic SR Ca leak in a CaMKII-dependent manner.
J Mol Cell Cardiol. 2014 Nov;76:94-105. doi: 10.1016/j.yjmcc.2014.08.016. Epub 2014 Aug 27.

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