Venta Amanda, Bick Johanna, Bechelli Jeremy
Department of Psychology, University of Houston, 4849 Calhoun Rd., Room 373, Houston, TX, 77204-6022, USA.
Department of Biological Sciences, Sam Houston State University, Huntsville, TX, USA.
Child Psychiatry Hum Dev. 2021 Apr;52(2):200-204. doi: 10.1007/s10578-021-01140-7. Epub 2021 Feb 22.
The COVID-19 pandemic exposed mothers to stress and social isolation during the pre- and post-natal periods. The deleterious effects of stress on both pregnant women and their infants are well documented, with research suggesting that effects are exacerbated by reduced social support. In this brief report, we summarize evidence linking stress and social isolation to negative outcomes for mothers and infants and present a conceptual model featuring inflammation as a driving mechanism. There is strong evidence that the coronavirus pandemic will affect mothers and infants through immune pathways that, in previous research, have been shown to link stress and social isolation during the pre- and post-natal periods with deficits in maternal mental health and infant well-being and development across developmental stages. We close with recommendations for novel research, policy changes, and integrated clinical care that can address these biological threats to infants and mothers while leveraging the anti-inflammatory effects of social support.
新冠疫情使母亲们在产前和产后面临压力和社会隔离。压力对孕妇及其婴儿的有害影响已有充分记录，研究表明，社会支持减少会加剧这些影响。在本简要报告中，我们总结了将压力和社会隔离与母婴负面结果联系起来的证据，并提出了一个以炎症为驱动机制的概念模型。有强有力的证据表明，冠状病毒大流行将通过免疫途径影响母婴，在先前的研究中，这些免疫途径已被证明将产前和产后期间的压力和社会隔离与母亲心理健康缺陷以及婴儿在各个发育阶段的福祉和发育联系起来。我们最后提出了新颖研究、政策变革和综合临床护理的建议，这些建议可以应对这些对婴儿和母亲的生物威胁，同时利用社会支持的抗炎作用。
