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述情障碍对抑郁症患者情绪刺激注意偏向的影响:一项眼动追踪研究

The Effect of Alexithymia on Attentional Bias Toward Emotional Stimuli in Depression: An Eye-Tracking Study.

作者信息

Bergman M Annemiek, Vissers Constance Th W M, Collard Rose M, van Eijndhoven Philip, Schene Aart H, Vrijsen Janna N

机构信息

Department of Psychiatry, Radboud University Medical Centre, Nijmegen, Netherlands.

Donders Institute for Brain, Cognition and Behavior, Donders Center for Medical Neuroscience, Nijmegen, Netherlands.

出版信息

Front Psychiatry. 2021 Feb 17;11:569946. doi: 10.3389/fpsyt.2020.569946. eCollection 2020.

DOI:10.3389/fpsyt.2020.569946
PMID:33679459
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7925401/
Abstract

Alexithymia-reflecting deficits in cognitive emotion processing-is highly prevalent in individuals with depressive disorders. Subsequently, mixed evidence for attentional bias is found in these individuals. Alexithymia may be a potential influencing factor for attentional bias in depression. In the current study, 83 currently depressed (CD) and 76 never-depressed (ND) controls completed an eye-tracker task consisting of valenced (non)-social pictures. Alexithymia scores were also included as a moderator as both a continuous and categorical measure (so high vs. low alexithymia). No group difference or moderating effect of alexithymia was found on attentional bias. Thus, alexithymic symptoms, included both dimensionally and categorically, may not influence biased attentional processing in depression compared to ND individuals. Thus, it is important to explore other potential explaining factors for the equivocal results found on biased attentional processing of emotional information in depression.

摘要

述情障碍反映了认知情绪加工方面的缺陷,在抑郁症患者中极为普遍。随后,在这些个体中发现了关于注意偏向的混合证据。述情障碍可能是抑郁症中注意偏向的一个潜在影响因素。在当前研究中,83名当前患有抑郁症(CD)的患者和76名从未患过抑郁症(ND)的对照者完成了一项由有情感色彩的(非)社交图片组成的眼动追踪任务。述情障碍得分也作为一个调节变量纳入研究,采用连续变量和分类变量两种测量方式(即高述情障碍与低述情障碍)。未发现述情障碍在注意偏向上存在组间差异或调节作用。因此,与ND个体相比,无论是从维度上还是分类上考虑,述情障碍症状可能都不会影响抑郁症患者的注意偏向加工。因此,探索其他可能解释抑郁症患者在情绪信息注意偏向加工上出现矛盾结果的潜在因素非常重要。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/de63/7925401/9459e2c03a7a/fpsyt-11-569946-g0003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/de63/7925401/8096737bab4a/fpsyt-11-569946-g0001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/de63/7925401/965182486a5b/fpsyt-11-569946-g0002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/de63/7925401/9459e2c03a7a/fpsyt-11-569946-g0003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/de63/7925401/8096737bab4a/fpsyt-11-569946-g0001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/de63/7925401/965182486a5b/fpsyt-11-569946-g0002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/de63/7925401/9459e2c03a7a/fpsyt-11-569946-g0003.jpg

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J Autism Dev Disord. 2021 Nov;51(11):4213-4226. doi: 10.1007/s10803-021-04880-6. Epub 2021 Jan 24.
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