相似文献
1
Correction to: Increase in excitability of hippocampal neurons during novelty-induced hyperlocomotion in dopamine-deficient mice.对《多巴胺缺乏小鼠在新奇诱导的运动亢进期间海马神经元兴奋性增加》的修正
Mol Brain. 2021 Mar 11;14(1):51. doi: 10.1186/s13041-021-00741-6.
2
Reversal of novelty-induced hyperlocomotion and hippocampal c-Fos expression in GluA1 knockout male mice by the mGluR2/3 agonist LY354740.谷氨酸受体 1 基因敲除雄性小鼠中海马 c-Fos 表达及新奇诱导过度活跃的反转由 mGluR2/3 激动剂 LY354740 介导。
Neuroscience. 2013 Oct 10;250:189-200. doi: 10.1016/j.neuroscience.2013.07.010. Epub 2013 Jul 15.
3
Attenuation of Novelty-Induced Hyperactivity of Mice by Cannabidiol and Hippocampal Inhibitory Chemogenetics.大麻二酚和海马抑制性化学遗传学对新奇诱导的小鼠多动的抑制作用
Front Pharmacol. 2019 Mar 29;10:309. doi: 10.3389/fphar.2019.00309. eCollection 2019.
4
Mice deficient in protein tyrosine phosphatase receptor type Z (PTPRZ) show reduced responsivity to methamphetamine despite an enhanced response to novelty.蛋白酪氨酸磷酸酯酶受体 Z 型（PTPRZ）缺失的小鼠对甲基苯丙胺的反应性降低，尽管对新奇事物的反应增强。
PLoS One. 2019 Aug 20;14(8):e0221205. doi: 10.1371/journal.pone.0221205. eCollection 2019.
5
Dopamine inhibits high-frequency stimulation-induced long-term potentiation of intrinsic excitability in CA1 hippocampal pyramidal neurons.多巴胺抑制海马CA1区锥体神经元高频刺激诱导的内在兴奋性长期增强。
Neurosignals. 2013;21(3-4):150-9. doi: 10.1159/000342435. Epub 2012 Sep 27.
6
Enhanced excitability of hippocampal mossy fibers and CA3 neurons under dietary zinc deficiency.膳食锌缺乏时海马苔藓纤维和CA3神经元兴奋性增强。
Epilepsy Res. 2005 Feb;63(2-3):77-84. doi: 10.1016/j.eplepsyres.2004.11.002.
7
Mice deficient for the extracellular matrix glycoprotein tenascin-r show physiological and structural hallmarks of increased hippocampal excitability, but no increased susceptibility to seizures in the pilocarpine model of epilepsy.缺乏细胞外基质糖蛋白腱生蛋白-R的小鼠表现出海马体兴奋性增加的生理和结构特征，但在匹鲁卡品癫痫模型中对癫痫发作的易感性并未增加。
Neuroscience. 2004;124(4):841-55. doi: 10.1016/j.neuroscience.2003.11.037.
8
Pseudoginsenoside-F11 inhibits methamphetamine-induced behaviors by regulating dopaminergic and GABAergic neurons in the nucleus accumbens.伪人参皂苷-F11通过调节伏隔核中的多巴胺能和γ-氨基丁酸能神经元来抑制甲基苯丙胺诱导的行为。
Psychopharmacology (Berl). 2016 Mar;233(5):831-40. doi: 10.1007/s00213-015-4159-8. Epub 2015 Dec 1.
9
Modification by the tissue plasminogen activator-plasmin system of morphine-induced dopamine release and hyperlocomotion, but not anti-nociceptive effect in mice.组织纤溶酶原激活物-纤溶酶系统对小鼠吗啡诱导的多巴胺释放和运动亢进的影响，但对其抗伤害感受作用无影响。
J Neurochem. 2005 Jun;93(5):1272-9. doi: 10.1111/j.1471-4159.2005.03117.x.
10
Inhibition by diazepam of ketamine-induced hyperlocomotion and dopamine turnover in mice.地西泮对小鼠氯胺酮诱导的运动亢进和多巴胺代谢的抑制作用。
Can J Anaesth. 1998 May;45(5 Pt 1):471-8. doi: 10.1007/BF03012584.
本文引用的文献
1
Increase in excitability of hippocampal neurons during novelty-induced hyperlocomotion in dopamine-deficient mice.在多巴胺缺乏的小鼠中，新奇诱导的过度活跃期间海马神经元兴奋性增加。
Mol Brain. 2020 Sep 18;13(1):126. doi: 10.1186/s13041-020-00664-8.
Zotero 插件