Retraction Note to: uPAR and cathepsin B downregulation induces apoptosis by targeting calcineurin A to BAD via Bcl-2 in glioma.
作者信息
Malla Rama Rao, Gopinath Sreelatha, Gondi Christopher S, Alapati Kiranmai, Dinh Dzung H, Tsung Andrew J, Rao Jasti S
机构信息
Department of Cancer Biology and Pharmacology, University of Illinois College of Medicine at Peoria, One Illini Drive, Peoria, IL, 61605, USA.
Department of Neurosurgery, University of Illinois College of Medicine at Peoria, One Illini Drive, Peoria, IL, 61605, USA.
出版信息
J Neurooncol. 2021 Apr;152(2):417. doi: 10.1007/s11060-021-03722-w.
Abstract
摘要
相似文献
1
Retraction Note to: uPAR and cathepsin B downregulation induces apoptosis by targeting calcineurin A to BAD via Bcl-2 in glioma.
J Neurooncol. 2021 Apr;152(2):417. doi: 10.1007/s11060-021-03722-w.
2
uPAR and cathepsin B downregulation induces apoptosis by targeting calcineurin A to BAD via Bcl-2 in glioma.uPAR 和组织蛋白酶 B 的下调通过 Bcl-2 将钙调神经磷酸酶 A 靶向 BAD，从而诱导神经胶质瘤细胞凋亡。
J Neurooncol. 2012 Mar;107(1):69-80. doi: 10.1007/s11060-011-0727-x. Epub 2011 Oct 2.
3
Downregulation of uPAR and cathepsin B induces apoptosis via regulation of Bcl-2 and Bax and inhibition of the PI3K/Akt pathway in gliomas.下调 uPAR 和组织蛋白酶 B 通过调节 Bcl-2 和 Bax 以及抑制 PI3K/Akt 通路诱导胶质瘤细胞凋亡。
PLoS One. 2010 Oct 29;5(10):e13731. doi: 10.1371/journal.pone.0013731.
4
Knockdown of cathepsin B and uPAR inhibits CD151 and α3β1 integrin-mediated cell adhesion and invasion in glioma.组织蛋白酶 B 和 uPAR 的敲低抑制了神经胶质瘤中 CD151 和 α3β1 整合素介导的细胞黏附和侵袭。
Mol Carcinog. 2013 Oct;52(10):777-90. doi: 10.1002/mc.21915. Epub 2012 Apr 11.
5
Co-depletion of cathepsin B and uPAR induces G0/G1 arrest in glioma via FOXO3a mediated p27 upregulation.组织蛋白酶 B 和 uPAR 的共耗竭通过 FOXO3a 介导的 p27 上调诱导胶质瘤 G0/G1 期阻滞。
PLoS One. 2010 Jul 22;5(7):e11668. doi: 10.1371/journal.pone.0011668.
6
uPAR and cathepsin B inhibition enhanced radiation-induced apoptosis in gliomainitiating cells.uPAR 和组织蛋白酶 B 抑制增强了致瘤性神经干细胞的放射诱导凋亡。
Neuro Oncol. 2012 Jun;14(6):745-60. doi: 10.1093/neuonc/nos088. Epub 2012 May 9.
7
Retraction: Downregulation of uPAR and Cathepsin B Induces Apoptosis via Regulation of Bcl-2 and Bax and Inhibition of the PI3K/Akt Pathway in Gliomas.撤稿声明：uPAR和组织蛋白酶B的下调通过调节Bcl-2和Bax以及抑制胶质瘤中的PI3K/Akt通路诱导细胞凋亡。
PLoS One. 2020 Jan 29;15(1):e0228688. doi: 10.1371/journal.pone.0228688. eCollection 2020.
8
Mechanism of p27 upregulation induced by downregulation of cathepsin B and uPAR in glioma.下调组织蛋白酶 B 和 uPAR 诱导胶质瘤中 p27 上调的机制。
Mol Oncol. 2011 Oct;5(5):426-37. doi: 10.1016/j.molonc.2011.07.004. Epub 2011 Jul 26.
9
RNA interference-mediated simultaneous down-regulation of urokinase-type plasminogen activator receptor and cathepsin B induces caspase-8-mediated apoptosis in SNB19 human glioma cells.RNA干扰介导的尿激酶型纤溶酶原激活物受体和组织蛋白酶B同时下调诱导SNB19人胶质瘤细胞中半胱天冬酶8介导的凋亡。
Mol Cancer Ther. 2006 Dec;5(12):3197-208. doi: 10.1158/1535-7163.MCT-05-0531.
10
MMP-9, uPAR and cathepsin B silencing downregulate integrins in human glioma xenograft cells in vitro and in vivo in nude mice.MMP-9、uPAR 和组织蛋白酶 B 的沉默下调了裸鼠体内外人神经胶质瘤异种移植细胞中的整合素。
PLoS One. 2010 Jul 15;5(7):e11583. doi: 10.1371/journal.pone.0011583.
Zotero 插件