Akhmedov Sh D, Gimrikh E O, Volkova T G, Idrisova E M, Pekarskiĭ V V
Kardiologiia. 1988 Feb;28(2):28-31.
Forty-nine patients, including 38 with documented bradysystolic sick-sinus syndrome (type I) and 11 with bradytachycardiac sick-sinus syndrome (type II) were studied. Follow-up of 24 patients with VVI stimulation (34 +/- 4 months) and 7 patients with AAI stimulation (23 +/- 1.2 months) demonstrated that VVI stimulation was associated with retrograde ventriculo-atrial conduction in 71% of patients, causing paroxysms of atrial fibrillation (5 patients, 4 of those having type I sick-sinus syndrome). Six patients developed permanent atrial fibrillation (including 5 with type I sick-sinus syndrome). Retrograde conduction slowed down the pulse rate because of ineffective ventricular response in 6 patients. Nine patients with retrograde conduction developed circulatory insufficiency.
对49例患者进行了研究，其中包括38例记录为缓慢性收缩性病态窦房结综合征（I型）和11例慢快综合征性病态窦房结综合征（II型）。对24例接受VVI刺激的患者（随访34±4个月）和7例接受AAI刺激的患者（随访23±1.2个月）的随访表明，71%的患者VVI刺激与逆行性室房传导有关，导致房颤发作（5例，其中4例为I型病态窦房结综合征）。6例患者发展为永久性房颤（包括5例I型病态窦房结综合征患者）。逆行传导因6例患者心室反应无效而使脉率减慢。9例有逆行传导的患者出现循环功能不全。
