Vandewalle H, Lacombe C, Lelièvre J C, Poirot C
Unité de Biorhéologie (C.N.R.S. UA 343), Université de Paris, France.
Int J Sports Med. 1988 Apr;9(2):104-7. doi: 10.1055/s-2007-1024988.
Ten healthy subjects performed two exercise sessions similar to an endurance training session for average athletes (1 cycling exercise at 85% of the maximal heart rate on a Monark cycle ergometer). In the first session, the subjects were not allowed to drink during exercise. During the second session, the same subjects performed the same exercise as during the first session but they drank a beverage volume equal to the weight loss induced by the first exercise session to verify the hypothesis that the compensation of sweat loss by drinking could attenuate the exercise-induced blood viscosity increase. Both protocols (with and without drinking) induced a significant increase of hematocrit and plasma viscosity. The whole blood viscosity increased at all shear rates but this increase was significant only for the exercise protocol without drinking. Blood thixotropic property, erythrocyte deformability, and erythrocyte aggregability remained unchanged after both exercise protocols. Hemoconcentration explained the increase of hematocrit and plasma viscosity. Hemoconcentration was probably the consequence of a filtration process through capillary leakage in addition to sweat loss, which could explain the partial effect of drinking in our study.
十名健康受试者进行了两次类似于普通运动员耐力训练课程的运动（在 Monark 自行车测力计上进行一次以最大心率 85%的骑行运动）。在第一次运动中，受试者在运动期间不允许饮水。在第二次运动中，相同的受试者进行了与第一次相同的运动，但他们饮用了与第一次运动导致的体重减轻量相等的饮料，以验证通过饮水补充汗液流失可以减弱运动引起的血液粘度增加这一假设。两种方案（饮水和不饮水）均导致血细胞比容和血浆粘度显著增加。全血粘度在所有剪切率下均增加，但仅在不饮水的运动方案中这种增加具有显著性。两种运动方案后血液触变性、红细胞变形性和红细胞聚集性均保持不变。血液浓缩解释了血细胞比容和血浆粘度的增加。除了汗液流失外，血液浓缩可能是通过毛细血管渗漏的过滤过程的结果，这可以解释我们研究中饮水的部分作用。
