Woo C C
J Manipulative Physiol Ther. 1988 Apr;11(2):118-23.
Early postmyelographic cauda equina syndrome in an asymptomatic young acromegalic is presented. The patient was asymptomatic for more than 1 yr despite myelographic evidence of acquired spinal stenosis at the L2-L4 level. Radiographic and/or myelographic findings should be clinically correlated. It is postulated that, in acromegaly, the combined simultaneous bony apposition-remodeling resorption mechanism is involved in the spine as platyspondyly, hyperostosis of spinous processes and vertebral scalloping to counteract soft tissue hyperplasia within the spinal canal; entrapment myelopathy, cauda equina syndrome and/or radiculopathy are ascribed to a soft tissue edematous mechanism superimposed on congenital or degenerative spinal stenosis, traumatic or postoperative spondylolisthesis, postmyelographic and/or postoperative arachnoiditis and a traumatic swollen or protrused intervertebral disc.
本文报道了一例无症状的年轻肢端肥大症患者在脊髓造影术后早期出现马尾综合征的病例。尽管脊髓造影显示L2 - L4水平存在后天性椎管狭窄，但该患者1年多来一直无症状。影像学和/或脊髓造影结果应与临床情况相关联。据推测，在肢端肥大症中，同时存在的骨增生 - 重塑吸收机制参与了脊柱病变，表现为椎体扁平、棘突骨质增生和椎体扇贝样改变，以对抗椎管内软组织增生；压迫性脊髓病、马尾综合征和/或神经根病归因于叠加在先天性或退行性椎管狭窄、创伤性或术后椎体滑脱、脊髓造影术后和/或术后蛛网膜炎以及创伤性肿胀或突出的椎间盘上的软组织水肿机制。
