Strakhov S N
Med Radiol (Mosk). 1988 Jun;33(6):8-14.
The author carried out a study of pulmonary hemodynamics in 155 patients aged 1 month to 15 yrs. old with acute and chronic pulmonary inflammatory diseases by radionuclide diagnostic, angiographic and other methods. It was established that angiological mechanisms of the pathogenesis of acute suppurative-destructive pneumonia were based on the reduction of the microcirculatory network of the pulmonary vessels with regional redistribution of the blood flow. Lesser circulatory rearrangement in chronic nonspecific pulmonary inflammatory diseases was characterized by regional hypoperfusion in the affected zone, a decrease in oxygen saturation of the blood, reduced blood flow volumes, and regional arterial pulmonary hypertension.
作者采用放射性核素诊断、血管造影及其他方法，对155例年龄在1个月至15岁之间患有急慢性肺部炎症性疾病的患者进行了肺血流动力学研究。结果表明，急性化脓性破坏性肺炎发病机制的血管学机制基于肺血管微循环网络的减少以及血流的区域重新分布。慢性非特异性肺部炎症性疾病中较小的循环重排表现为受累区域的局部灌注不足、血液氧饱和度降低、血流量减少以及局部动脉性肺动脉高压。
