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[视力丧失作为急性胰腺炎的一种并发症]

[Loss of vision as a complication of acute pancreatitis].

作者信息

Flaggl E, Heer M, Hany A, Branda L

机构信息

Medizinische Klinik und Augenklinik, Kantonsspital Winterthur.

出版信息

Schweiz Med Wochenschr. 1988 May 14;118(19):722-5.

PMID:3387968
Abstract

A report is presented on two patients with severe visual loss and scotoma following an episode of alcohol-induced pancreatitis. A 35-year-old man with pancreatitis developed visual loss in both eyes. Ophthalmoscopic examination revealed extensive ischemic infarcts with bilateral hemorrhage and cotton-wool spots, a clinical picture similar to that of (post-traumatic) Purtscher's retinopathy. Goldmann visual fields demonstrated paracentral scotomas. Fundoscopic lesions had disappeared 3 months after the acute event and visual acuity improved gradually from 0.1 to 1.0. In a 36-year-old man, cerebral infarction was established by CT as a possible cause of visual loss. CSF examination revealed Sudan-III positive material suggesting cerebral fat embolism as the cause of cerebral infarcts. Visual fields showed central scotomas. During the 4-year follow-up period there was a gradual improvement in visual acuity.

摘要

本文报告了两名在酒精性胰腺炎发作后出现严重视力丧失和暗点的患者。一名35岁患有胰腺炎的男性双眼出现视力丧失。眼底检查发现广泛的缺血性梗死伴双侧出血和棉絮斑,临床表现类似于(创伤后)普尔夏视网膜病变。戈德曼视野检查显示旁中心暗点。急性事件发生3个月后眼底病变消失,视力从0.1逐渐提高到1.0。一名36岁男性经CT检查确诊为脑梗死,可能是视力丧失的原因。脑脊液检查发现苏丹III阳性物质,提示脑脂肪栓塞是脑梗死的原因。视野检查显示中心暗点。在4年的随访期内,视力逐渐改善。

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