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How Do We Know What We Are Missing? Loss of Signaling through CD148 Drives Fibroblast Activation in Pulmonary Fibrosis.

作者信息

Redente Elizabeth F

机构信息

Department of Pediatrics National Jewish Health Denver, Colorado and.

Department of Medicine University of Colorado School of Medicine Aurora, Colorado.

出版信息

Am J Respir Crit Care Med. 2021 Aug 1;204(3):249-251. doi: 10.1164/rccm.202103-0737ED.

Abstract
摘要

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本文引用的文献

1
CD148 Deficiency in Fibroblasts Promotes the Development of Pulmonary Fibrosis.
Am J Respir Crit Care Med. 2021 Aug 1;204(3):312-325. doi: 10.1164/rccm.202008-3100OC.
2
Syndecan receptors: pericellular regulators in development and inflammatory disease.
Open Biol. 2021 Feb;11(2):200377. doi: 10.1098/rsob.200377. Epub 2021 Feb 10.
3
Persistent, Progressive Pulmonary Fibrosis and Epithelial Remodeling in Mice.
Am J Respir Cell Mol Biol. 2021 Jun;64(6):669-676. doi: 10.1165/rcmb.2020-0542MA.
4
Single-cell RNA-seq reveals ectopic and aberrant lung-resident cell populations in idiopathic pulmonary fibrosis.
Sci Adv. 2020 Jul 8;6(28):eaba1983. doi: 10.1126/sciadv.aba1983. eCollection 2020 Jul.
5
Single-cell RNA sequencing reveals profibrotic roles of distinct epithelial and mesenchymal lineages in pulmonary fibrosis.
Sci Adv. 2020 Jul 8;6(28):eaba1972. doi: 10.1126/sciadv.aba1972. eCollection 2020 Jul.
6
Syndecan-4 Inhibits the Development of Pulmonary Fibrosis by Attenuating TGF-β Signaling.
Int J Mol Sci. 2019 Oct 9;20(20):4989. doi: 10.3390/ijms20204989.
7
The therapy of idiopathic pulmonary fibrosis: what is next?
Eur Respir Rev. 2019 Sep 4;28(153). doi: 10.1183/16000617.0021-2019. Print 2019 Sep 30.
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Idiopathic pulmonary fibrosis: lessons from clinical trials over the past 25 years.
Eur Respir J. 2017 Oct 26;50(4). doi: 10.1183/13993003.01209-2017. Print 2017 Oct.

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